Strain dependent and invariant features of transgenic mice expressing Alzheimer amyloid precursor proteins. (Q48291104)

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9 December 2019

review article

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title

Strain dependent and invariant features of transgenic mice expressing Alzheimer amyloid precursor proteins (English)

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:9932419%20AND%20SRC:MED&resulttype=core&format=json

9 December 2019

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K Hsiao

1

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9 December 2019

publication date

1 January 1998

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Progress in Brain Research

9 December 2019

published in

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:9932419%20AND%20SRC:MED&resulttype=core&format=json

9 December 2019

volume

117

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9 December 2019

page(s)

335-341

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Lack of apolipoprotein E dramatically reduces amyloid beta-peptide deposition

9 December 2019

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Accelerated amyloid deposition in the brains of transgenic mice coexpressing mutant presenilin 1 and amyloid precursor proteins

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Mutation of the beta-amyloid precursor protein in familial Alzheimer's disease increases beta-protein production

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Increased amyloid-beta42(43) in brains of mice expressing mutant presenilin 1

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Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein

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Early Alzheimer disease-like histopathology increases in frequency with age in mice transgenic for beta-APP751.

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Accelerated Alzheimer-type phenotype in transgenic mice carrying both mutant amyloid precursor protein and presenilin 1 transgenes

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Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

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APPSw transgenic mice develop age-related A beta deposits and neuropil abnormalities, but no neuronal loss in CA1.

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Amyloid precursor protein processing and A beta42 deposition in a transgenic mouse model of Alzheimer disease

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Deposition of beta/A4 immunoreactivity and neuronal pathology in transgenic mice expressing the carboxyl-terminal fragment of the Alzheimer amyloid precursor in the brain

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The Alzheimer's A beta peptide induces neurodegeneration and apoptotic cell death in transgenic mice

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Expression in brain of amyloid precursor protein mutated in the alpha-secretase site causes disturbed behavior, neuronal degeneration and premature death in transgenic mice.

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Impaired learning and LTP in mice expressing the carboxy terminus of the Alzheimer amyloid precursor protein

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Transgenic mice expressing APP-C100 in the brain.

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Age-dependent neuronal and synaptic degeneration in mice transgenic for the C terminus of the amyloid precursor protein.

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Two amyloid precursor protein transgenic mouse models with Alzheimer disease-like pathology

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An increased percentage of long amyloid beta protein secreted by familial amyloid beta protein precursor (beta APP717) mutants.

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Formation of beta-amyloid protein deposits in brains of transgenic mice

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7 January 2021

Amyloid-β Deposition in Alzheimer Transgenic Mice Is Associated with Oxidative Stress

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Increased susceptibility to ischemic brain damage in transgenic mice overexpressing the amyloid precursor protein.

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7 January 2021

10.1016/S0079-6123(08)64026-1

Identifiers

DOI

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:9932419%20AND%20SRC:MED&resulttype=core&format=json

9 December 2019

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