Poly (ADP-ribose) polymerase induction is an early signal of apoptosis in human neuroblastoma. (Q50512856)

14 March 2018

title

Poly (ADP-ribose) polymerase induction is an early signal of apoptosis in human neuroblastoma. (English)

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14 March 2018

author name string

Bursztajn S

1

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14 March 2018

Feng JJ

2

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14 March 2018

Berman SA

3

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14 March 2018

Nanda A

4

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14 March 2018

publication date

1 March 2000

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14 March 2018

published in

Molecular Brain Research

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14 March 2018

volume

76

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14 March 2018

issue

2

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14 March 2018

page(s)

363-376

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14 March 2018

Externalization of phosphatidylserine may not be an early signal of apoptosis in neuronal cells, but only the phosphatidylserine-displaying apoptotic cells are phagocytosed by microglia

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Mechanisms of delayed cell death following hypoxic-ischemic injury in the immature rat: evidence for apoptosis during selective neuronal loss

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Apoptosis and Its Modulation in Human Promyelocytic HL-60 Cells Treated with DNA Topoisomerase I and II Inhibitors

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Expression of the poly(ADP-ribose) polymerase gene following natural and induced DNA strand breakage and effect of hyperexpression on DNA repair.

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Acetylcholine receptor clusters are associated with nuclei in rat myotubes

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Commitment to cell death measured by loss of clonogenicity is separable from the appearance of apoptotic markers

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Key morphological features of apoptosis may occur in the absence of internucleosomal DNA fragmentation

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A biochemical hallmark of apoptosis: internucleosomal degradation of the genome

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DNA strand breaks induced by sustained glutamate excitotoxicity in primary neuronal cultures.

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Activation of a caspase 3-related cysteine protease is required for glutamate-mediated apoptosis of cultured cerebellar granule neurons

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Induction of apoptosis in chronic lymphocytic leukemia cells and its prevention by phorbol ester

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The apoptosis-necrosis paradox. Apoptogenic proteases activated after mitochondrial permeability transition determine the mode of cell death.

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Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4

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Cleavage of poly(ADP-ribose) polymerase by a proteinase with properties like ICE

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Studies of the lamin proteinase reveal multiple parallel biochemical pathways during apoptotic execution

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Inhibition of Ced-3/ICE-related proteases does not prevent cell death induced by oncogenes, DNA damage, or the Bcl-2 homologue Bak

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Overproduction of the poly(ADP-ribose) polymerase DNA-binding domain blocks alkylation-induced DNA repair synthesis in mammalian cells

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Activation and cleavage of caspase-3 in apoptosis induced by experimental cerebral ischemia.

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Cell-free apoptosis in Xenopus egg extracts: inhibition by Bcl-2 and requirement for an organelle fraction enriched in mitochondria.

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Identification and inhibition of the ICE/CED-3 protease necessary for mammalian apoptosis

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ICE/CED3-like proteases as therapeutic targets for the control of inappropriate apoptosis

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Cell death during development of the nervous system

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The CED-3/ICE-like protease Mch2 is activated during apoptosis and cleaves the death substrate lamin A

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7 January 2021

Separate pools of endonuclease activity are responsible for internucleosomal and high molecular mass DNA fragmentation during apoptosis.

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DNA fragmentation during apoptosis is caused by frequent single-strand cuts

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Evidence for apoptotic cell death in Huntington disease and excitotoxic animal models.

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Characterization of CPP32-like protease activity following apoptotic challenge in SH-SY5Y neuroblastoma cells

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A simple and rapid method for detection of apoptosis in human cells

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7 January 2021

Dual function for poly(ADP-ribose) synthesis in response to DNA strand breakage

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Mechanisms and genes of cellular suicide

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7 January 2021

Mechanisms of apoptosis: integration of genetic, biochemical, and cellular indicators

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7 January 2021

Yama/CPP32 beta, a mammalian homolog of CED-3, is a CrmA-inhibitable protease that cleaves the death substrate poly(ADP-ribose) polymerase

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7 January 2021

Apoptosis in the pathogenesis and treatment of disease

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7 January 2021

Endonuclease activation following focal ischemic injury in the rat brain

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7 January 2021

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7 January 2021

Increased sensitivity to DNA-alkylating agents in CHO mutants with decreased poly(ADP-ribose) polymerase activity

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7 January 2021

Activation of CPP32-like caspases contributes to neuronal apoptosis and neurological dysfunction after traumatic brain injury.

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7 January 2021

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Identifiers

DOI

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14 March 2018

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14 March 2018