Neuroprotective effects of resveratrol in an MPTP mouse model of Parkinson's-like disease: possible role of SOCS-1 in reducing pro-inflammatory responses. (Q50910086)

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scientific article published on 13 June 2013

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English	Neuroprotective effects of resveratrol in an MPTP mouse model of Parkinson's-like disease: possible role of SOCS-1 in reducing pro-inflammatory responses.	scientific article published on 13 June 2013

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scholarly article

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3 March 2020

Neuroprotective effects of resveratrol in an MPTP mouse model of Parkinson's-like disease: possible role of SOCS-1 in reducing pro-inflammatory responses (English)

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3 March 2020

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Parkinson's disease

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based on heuristic

inferred from title

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based on heuristic

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Dario Domenico Lofrumento

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3 March 2020

Giuseppe Nicolardi

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3 March 2020

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Velia La Pesa

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Antonia Cianciulli

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Antonia Cianciulli

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Francesco De Nuccio

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Francesco De Nuccio

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Vito Carofiglio

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Teresa Dragone

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Maria A Panaro

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publication date

13 June 2013

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Innate Immunity

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3 March 2020

Levodopa for the treatment of Parkinson's disease

1 reference

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Modeling Parkinson's disease in rats: an evaluation of 6-OHDA lesions of the nigrostriatal pathway

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Brain dopamine and the syndromes of Parkinson and Huntington. Clinical, morphological and neurochemical correlations

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Parkinson's disease: mechanisms and models

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Reactive microglia are positive for HLA-DR in the substantia nigra of Parkinson's and Alzheimer's disease brains

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Neuroinflammatory mechanisms in Parkinson's disease: potential environmental triggers, pathways, and targets for early therapeutic intervention

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Microglia in health and disease

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Tumor necrosis factor-alpha (TNF-alpha) increases both in the brain and in the cerebrospinal fluid from parkinsonian patients.

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Inflammatory regulators in Parkinson's disease: iNOS, lipocortin-1, and cyclooxygenases-1 and -2.

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Expression and functional significance of SOCS-1 and SOCS-3 in astrocytes

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Biological effects of resveratrol

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Resveratrol and its analogs: defense against cancer, coronary disease and neurodegenerative maladies or just a fad?

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Dietary supplementation with resveratrol reduces plaque pathology in a transgenic model of Alzheimer's disease

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Resveratrol promotes clearance of Alzheimer's disease amyloid-beta peptides.

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AMP-activated protein kinase signaling activation by resveratrol modulates amyloid-beta peptide metabolism.

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Resveratrol protects dopaminergic neurons in midbrain slice culture from multiple insults.

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Dietary oxyresveratrol prevents parkinsonian mimetic 6-hydroxydopamine neurotoxicity

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Anti-inflammatory activities of resveratrol in the brain: role of resveratrol in microglial activation

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MPTP-induced neuroinflammation increases the expression of pro-inflammatory cytokines and their receptors in mouse brain.

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Resveratrol, a red wine polyphenol, protects dopaminergic neurons in MPTP-treated mice

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21 January 2018

Resveratrol attenuates 6-hydroxydopamine-induced oxidative damage and dopamine depletion in rat model of Parkinson's disease.

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Neuroprotective effect of resveratrol on 6-OHDA-induced Parkinson's disease in rats

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Fate of nigrostriatal neurons in young mature mice given 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine: a neurochemical and morphological reassessment.

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Intrastriatal infusion of the Parkinsonian neurotoxin, MPP(+), induces damage of striatal cell nuclei in Sprague-Dawley rats

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Protocol for the MPTP mouse model of Parkinson's disease

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A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

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Resveratrol protects against global cerebral ischemic injury in gerbils

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Transgenic expression and activation of PGC-1α protect dopaminergic neurons in the MPTP mouse model of Parkinson's disease

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Cytokines and related receptor-mediated signaling pathways

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Resveratrol inhibits phorbol ester-induced expression of COX-2 and activation of NF-kappaB in mouse skin by blocking IkappaB kinase activity

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Resveratrol analogues as selective cyclooxygenase-2 inhibitors: synthesis and structure-activity relationship

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Neuroinflammation mediated by IL-1beta increases susceptibility of dopamine neurons to degeneration in an animal model of Parkinson's disease

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