Role of p38 mitogen-activated protein kinase in cardiac myocyte secretion of the inflammatory cytokine TNF-alpha. (Q51360555)

4 April 2018

title

Role of p38 mitogen-activated protein kinase in cardiac myocyte secretion of the inflammatory cytokine TNF-alpha. (English)

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4 April 2018

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C Ballard-Croft

1

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4 April 2018

D J White

2

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4 April 2018

D L Maass

3

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4 April 2018

D P Hybki

4

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4 April 2018

J W Horton

5

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publication date

1 May 2001

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4 April 2018

American Journal of Physiology Heart and Circulatory Physiology

published in

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4 April 2018

volume

280

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4 April 2018

issue

5

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page(s)

H1970-81

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4 April 2018

Efficacy and safety of monoclonal antibody to human tumor necrosis factor alpha in patients with sepsis syndrome. A randomized, controlled, double-blind, multicenter clinical trial. TNF-alpha MAb Sepsis Study Group.

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Therapeutic strategies to reduce TNF-alpha mediated cardiac contractile depression following ischemia and reperfusion

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Tumor necrosis factor-alpha and interleukin-1beta synergistically depress human myocardial function

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SB 203580 is a specific inhibitor of a MAP kinase homologue which is stimulated by cellular stresses and interleukin-1

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Transcriptional regulation by MAP kinases

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Effect of stress and trauma on bacterial translocation from the gut.

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Protective effect of 55- but not 75-kD soluble tumor necrosis factor receptor-immunoglobulin G fusion proteins in an animal model of gram-negative sepsis

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Induction of cyclooxygenase-2 by the activated MEKK1 --> SEK1/MKK4 --> p38 mitogen-activated protein kinase pathway

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Nitric oxide modulation of TNF-alpha-induced cardiac contractile dysfunction is concentration dependent.

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P38 Mitogen Activated Protein Kinase Regulates Endothelial VCAM-1 Expression at the Post-transcriptional Level

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Hypoxia and hypoxia/reoxygenation activate p65PAK, p38 mitogen-activated protein kinase (MAPK), and stress-activated protein kinase (SAPK) in cultured rat cardiac myocytes

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ERKs and p38 kinase phosphorylate p53 protein at serine 15 in response to UV radiation

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Jun N-terminal kinase/stress-activated protein kinase (JNK/SAPK) is required for lipopolysaccharide stimulation of tumor necrosis factor alpha (TNF-alpha) translation: glucocorticoids inhibit TNF-alpha translation by blocking JNK/SAPK.

21 January 2018

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21 January 2018

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Cellular basis for the negative inotropic effects of tumor necrosis factor-alpha in the adult mammalian heart

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Sphingosine Mediates the Immediate Negative Inotropic Effects of Tumor Necrosis Factor-α in the Adult Mammalian Cardiac Myocyte

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10.1152/AJPHEART.2001.280.5.H1970

21 January 2018

Identifiers

DOI

1 reference

4 April 2018

release_i3wak7qsrzdjxgiejy7cyhltr4

Fatcat ID

1 reference

Fatcat

https://api.fatcat.wiki/v0/release/i3wak7qsrzdjxgiejy7cyhltr4

mapped directly with Wikidata item

24 November 2022

based on heuristic

PubMed ID

11299196

1 reference

4 April 2018