The Influence of Radiotherapy on AIM2 Inflammasome in Radiation Pneumonitis. (Q51571232)

7 April 2018

title

The Influence of Radiotherapy on AIM2 Inflammasome in Radiation Pneumonitis. (English)

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7 April 2018

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Qianyu Zhang

1

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7 April 2018

Qinyong Hu

2

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7 April 2018

Yuxin Chu

3

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7 April 2018

Bin Xu

4

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7 April 2018

Qibin Song

5

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7 April 2018

publication date

15 August 2016

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7 April 2018

published in

Inflammation

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7 April 2018

volume

39

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7 April 2018

issue

5

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7 April 2018

page(s)

1827-1834

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7 April 2018

Transforming growth factor-beta-1 is a serum biomarker of radiation-induced pneumonitis in esophageal cancer patients treated with thoracic radiotherapy: preliminary results of a prospective study

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AIM2 activates the inflammasome and cell death in response to cytoplasmic DNA

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Increased expression of MyD88 and association with paclitaxel resistance in breast cancer

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Genistein alleviates radiation-induced pneumonitis by depressing Ape1/Ref-1 expression to down-regulate inflammatory cytokines

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Autophagy proteins regulate innate immune responses by inhibiting the release of mitochondrial DNA mediated by the NALP3 inflammasome

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Oxidized mitochondrial DNA activates the NLRP3 inflammasome during apoptosis

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Inhibition of Radiation-Induced Oxidative Damage in the Lung Tissue: May Acetylsalicylic Acid Have a Positive Role?

21 January 2018

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PubMed

https://pubmed.ncbi.nlm.nih.gov/27525422

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1007/S10753-016-0419-Y

1 reference

7 April 2018

1 reference

7 April 2018