The relationship between mucosal cyclooxygenase-2 (COX-2) expression and experimental radiation-induced mucositis. (Q51830987)

13 April 2018

title

The relationship between mucosal cyclooxygenase-2 (COX-2) expression and experimental radiation-induced mucositis. (English)

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13 April 2018

author name string

Sonis ST

1

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13 April 2018

O'Donnell KE

2

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13 April 2018

Popat R

3

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13 April 2018

Bragdon C

4

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13 April 2018

Phelan S

5

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13 April 2018

Cocks D

6

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13 April 2018

Epstein JB

7

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13 April 2018

language of work or name

English

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publication date

1 February 2004

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13 April 2018

published in

Oral Oncology

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13 April 2018

volume

40

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13 April 2018

issue

2

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13 April 2018

page(s)

170-176

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13 April 2018

Mucositis as a biological process: a new hypothesis for the development of chemotherapy-induced stomatotoxicity.

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Defining mechanisms of action of interleukin-11 on the progression of radiation-induced oral mucositis in hamsters

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Transcriptional roles of nuclear factor kappa B and nuclear factor-interleukin-6 in the tumor necrosis factor alpha-dependent induction of cyclooxygenase-2 in MC3T3-E1 cells

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Rapid and transient induction of cyclo-oxygenase 2 by epidermal growth factor in human amnion-derived WISH cells

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Transforming growth factor-beta1 enhances Ha-ras-induced expression of cyclooxygenase-2 in intestinal epithelial cells via stabilization of mRNA.

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Differential regulation of cyclooxygenases 1 and 2 by interleukin-1 beta, tumor necrosis factor-alpha, and transforming growth factor-beta 1 in human lung fibroblasts.

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Tumor necrosis factor-alpha promotes sustained cyclooxygenase-2 expression: attenuation by dexamethasone and NSAIDs

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Radiation induces upregulation of cyclooxygenase-2 (COX-2) protein in PC-3 cells

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Retinoic acid-inducible gene-I is induced in endothelial cells by LPS and regulates expression of COX-2.

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Cyclo-oxygenase-2: pharmacology, physiology, biochemistry and relevance to NSAID therapy

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Identifiers

DOI

1 reference

13 April 2018

PubMed ID

14693241

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13 April 2018