Sarcoplasmic reticulum calcium defect in Ras-induced hypertrophic cardiomyopathy heart. (Q52551784)

3 May 2018

title

Sarcoplasmic reticulum calcium defect in Ras-induced hypertrophic cardiomyopathy heart. (English)

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3 May 2018

hypertrophic cardiomyopathy

main subject

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11

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author name string

Meizi Zheng

1

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Keith Dilly

2

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Jader Dos Santos Cruz

3

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Manxiang Li

4

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Yusu Gu

5

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Jeanine A Ursitti

6

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Ju Chen

7

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John Ross

8

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Kenneth R Chien

9

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Jonathan W Lederer

10

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publication date

11 September 2003

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3 May 2018

American Journal of Physiology Heart and Circulatory Physiology

published in

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volume

286

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page(s)

H424-33

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issue

1

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A Ras-dependent pathway regulates RNA polymerase II phosphorylation in cardiac myocytes: implications for cardiac hypertrophy

cites work

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Biophysical interaction between phospholamban and protein phosphatase 1 regulatory subunit GM.

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Protein phosphatase activity is increased in a rat model of long-term beta-adrenergic stimulation

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Familial hypertrophic cardiomyopathy: from mutations to functional defects

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The MEK1-ERK1/2 signaling pathway promotes compensated cardiac hypertrophy in transgenic mice

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Selective requirement of myosin light chain 2v in embryonic heart function

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Heart failure after myocardial infarction: altered excitation-contraction coupling.

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Ras-dependent pathways induce obstructive hypertrophy in echo-selected transgenic mice

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Ras reduces L-type calcium channel current in cardiac myocytes. Corrective effects of L-channels and SERCA2 on [Ca(2+)](i) regulation and cell morphology

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The Raf-MEK-ERK cascade represents a common pathway for alteration of intracellular calcium by Ras and protein kinase C in cardiac myocytes.

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The low molecular weight GTPase Rho regulates myofibril formation and organization in neonatal rat ventricular myocytes. Involvement of Rho kinase

21 January 2018

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Diminished basal phosphorylation level of phospholamban in the postinfarction remodeled rat ventricle: role of beta-adrenergic pathway, G(i) protein, phosphodiesterase, and phosphatases

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Ventricular expression of a MLC-2v-ras fusion gene induces cardiac hypertrophy and selective diastolic dysfunction in transgenic mice.

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Expression of cellular oncogenes in the myocardium during the developmental stage and pressure-overloaded hypertrophy of the rat heart.

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Guanine nucleotide exchange factor-like factor (Rlf) induces gene expression and potentiates alpha 1-adrenergic receptor-induced transcriptional responses in neonatal rat ventricular myocytes

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Segregation of atrial-specific and inducible expression of an atrial natriuretic factor transgene in an in vivo murine model of cardiac hypertrophy

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Rho is required for Galphaq and alpha1-adrenergic receptor signaling in cardiomyocytes. Dissociation of Ras and Rho pathways

21 January 2018

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Ca2+ flux through promiscuous cardiac Na+ channels: slip-mode conductance.

21 January 2018

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Calcium sparks and excitation-contraction coupling in phospholamban-deficient mouse ventricular myocytes.

21 January 2018

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Signaling in myocardial hypertrophy: life after calcineurin?

21 January 2018

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Targeted DNA recombination in vivo using an adenovirus carrying the cre recombinase gene

21 January 2018

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Transgenic expression of replication-restricted enteroviral genomes in heart muscle induces defective excitation-contraction coupling and dilated cardiomyopathy

21 January 2018

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21 January 2018

Identifiers

DOI

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3 May 2018

PubMed ID

12969887

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3 May 2018