Proteomic analysis of steroid-triggered autophagic programmed cell death during Drosophila development. (Q52676250)

5 May 2018

title

Proteomic analysis of steroid-triggered autophagic programmed cell death during Drosophila development. (English)

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Don L DeVoe

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author name string

Martin DN

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Dutta S

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Chen J

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Rudnick P

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Cranford J

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Kantartzis S

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Lee C

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Baehrecke EH

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publication date

26 January 2007

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Cell Death & Differentiation

published in

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volume

14

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issue

5

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page(s)

916-923

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https://scigraph.springernature.com/pub.10.1038/sj.cdd.4402098

exact match

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Caspases function in autophagic programmed cell death in Drosophila

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The role of apoptosis in creating and maintaining luminal space within normal and oncogene-expressing mammary acini.

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Role of Bcl-2 family proteins in a non-apoptotic programmed cell death dependent on autophagy genes

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Drosophila ultraspiracle modulates ecdysone receptor function via heterodimer formation

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The Drosophila beta FTZ-F1 orphan nuclear receptor provides competence for stage-specific responses to the steroid hormone ecdysone.

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E93 directs steroid-triggered programmed cell death in Drosophila

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Genome-wide analyses of steroid- and radiation-triggered programmed cell death in Drosophila.

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The Drosophila melanogaster Apaf-1 homologue ARK is required for most, but not all, programmed cell death

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The Drosophila caspase Ice is important for many apoptotic cell deaths and for spermatid individualization, a nonapoptotic process

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Highwire regulates presynaptic BMP signaling essential for synaptic growth.

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The Salvador partner Hippo promotes apoptosis and cell-cycle exit in Drosophila.

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7 January 2021

based on heuristic

The Drosophila insulin receptor homolog: a gene essential for embryonic development encodes two receptor isoforms with different signaling potential.

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inferred from DOI database lookup

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The Drosophila tumor suppressor gene warts encodes a homolog of human myotonic dystrophy kinase and is required for the control of cell shape and proliferation

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4402098

inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1038/SJ.CDD.4402098

1 reference

5 May 2018

PubMed ID

17256009

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5 May 2018