Accumulative copy number increase of MET drives tumor development and histological progression in a subset of ovarian clear-cell adenocarcinomas. (Q53280611)

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scientific article published on 7 October 2011

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English	Accumulative copy number increase of MET drives tumor development and histological progression in a subset of ovarian clear-cell adenocarcinomas.	scientific article published on 7 October 2011

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scholarly article

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Europe PubMed Central

12 May 2018

http://europepmc.org/abstract/MED/21983935

Accumulative copy number increase of MET drives tumor development and histological progression in a subset of ovarian clear-cell adenocarcinomas. (English)

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Europe PubMed Central

12 May 2018

http://europepmc.org/abstract/MED/21983935

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Hitoshi Tsuda

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http://europepmc.org/abstract/MED/21983935

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Masashi Takano

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12 May 2018

http://europepmc.org/abstract/MED/21983935

Osamu Matsubara

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object named as

Osamu Matsubara

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http://europepmc.org/abstract/MED/21983935

author name string

Sohei Yamamoto

1

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Europe PubMed Central

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http://europepmc.org/abstract/MED/21983935

Kosuke Miyai

3

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Europe PubMed Central

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http://europepmc.org/abstract/MED/21983935

Seiichi Tamai

5

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http://europepmc.org/abstract/MED/21983935

publication date

7 October 2011

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http://europepmc.org/abstract/MED/21983935

Modern Pathology

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25

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http://europepmc.org/abstract/MED/21983935

1

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122-130

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12 May 2018

http://europepmc.org/abstract/MED/21983935

https://scigraph.springernature.com/pub.10.1038/modpathol.2011.143

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c-Met: structure, functions and potential for therapeutic inhibition

1 reference

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Targeting the c-Met signaling pathway in cancer

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Met, metastasis, motility and more

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Novel mutations of the MET proto-oncogene in papillary renal carcinomas.

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Frequent amplification of the c-met gene in scirrhous type stomach cancer.

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Homozygous deletions and chromosome amplifications in human lung carcinomas revealed by single nucleotide polymorphism array analysis

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MET amplification occurs with or without T790M mutations in EGFR mutant lung tumors with acquired resistance to gefitinib or erlotinib

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Do clear cell ovarian carcinomas have poorer prognosis compared to other epithelial cell types? A study of 1411 clear cell ovarian cancers

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Long-term follow-up and prognostic factor analysis in clear cell adenocarcinoma of the ovary

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Clear cell carcinoma of the ovary: a retrospective multicentre experience of 254 patients with complete surgical staging.

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Cumulative alterations of p27-related cell-cycle regulators in the development of endometriosis-associated ovarian clear cell adenocarcinoma.

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The origin and pathogenesis of epithelial ovarian cancer: a proposed unifying theory

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Ovarian clear cell adenocarcinoma: a continuing enigma

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Assessment of methods for tissue-based detection of the HER-2/neu alteration in human breast cancer: a direct comparison of fluorescence in situ hybridization and immunohistochemistry

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Comparison of c-met expression in ovarian epithelial tumors and normal epithelia of the female reproductive tract by quantitative laser scan microscopy

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An orally available small-molecule inhibitor of c-Met, PF-2341066, reduces tumor burden and metastasis in a preclinical model of ovarian cancer metastasis

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Functional expression and mutations of c-Met and its therapeutic inhibition with SU11274 and small interfering RNA in non-small cell lung cancer.

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Expression and mutational analysis of MET in human solid cancers

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MET amplification leads to gefitinib resistance in lung cancer by activating ERBB3 signaling

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Amplification of MET may identify a subset of cancers with extreme sensitivity to the selective tyrosine kinase inhibitor PHA-665752

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NK4 (HGF-antagonist/angiogenesis inhibitor) in cancer biology and therapeutics

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AMG 102, a fully human anti-hepatocyte growth factor/scatter factor neutralizing antibody, enhances the efficacy of temozolomide or docetaxel in U-87 MG cells and xenografts

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Clear cell adenocarcinoma with a component of poorly differentiated histology: a poor prognostic subgroup of ovarian clear cell adenocarcinoma

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Evaluation of the tyrosine kinase domain of the Met proto-oncogene in sporadic ovarian carcinomas*

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Amplification of growth factor receptor genes and DNA ploidy pattern in the progression of gastric cancer

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Genomic amplification of MET with boundaries within fragile site FRA7G and upregulation of MET pathways in esophageal adenocarcinoma

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Gene amplification and protein overexpression of MET are common events in ovarian clear-cell adenocarcinoma: their roles in tumor progression and prognostication of the patient

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Clear cell epithelial ovarian cancer (mesonephroid): bad prognosis only in early stages

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Molecular genetic evidence that endometriosis is a precursor of ovarian cancer

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Ovarian atypical endometriosis: its close association with malignant epithelial tumours

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Expression of platelet-derived growth factors and their receptors in ovarian clear-cell carcinoma and its putative precursors

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Immunopathogenesis of pelvic endometriosis: role of hepatocyte growth factor, macrophages and ovarian steroids.

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7 January 2021

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c-Met overexpression is a prognostic factor in ovarian cancer and an effective target for inhibition of peritoneal dissemination and invasion

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Reversion of human glioblastoma malignancy by U1 small nuclear RNA/ribozyme targeting of scatter factor/hepatocyte growth factor and c-met expression

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Identifiers

10.1038/MODPATHOL.2011.143

1 reference

Europe PubMed Central

12 May 2018

http://europepmc.org/abstract/MED/21983935

1 reference

Europe PubMed Central

12 May 2018

http://europepmc.org/abstract/MED/21983935

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