Caspase-8 Mediates Amyloid-β-induced Apoptosis in Differentiated PC12 Cells. (Q53621234)

16 May 2018

title

Caspase-8 Mediates Amyloid-β-induced Apoptosis in Differentiated PC12 Cells. (English)

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16 May 2018

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Min-cai Qian

1

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16 May 2018

Jing Liu

2

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16 May 2018

Jia-shu Yao

3

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16 May 2018

Wei-min Wang

4

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16 May 2018

Jian-hong Yang

5

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16 May 2018

Li-li Wei

6

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16 May 2018

Yue-di Shen

7

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16 May 2018

Wei Chen

8

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16 May 2018

publication date

3 February 2015

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16 May 2018

Journal of Molecular Neuroscience

published in

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16 May 2018

volume

56

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16 May 2018

issue

2

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16 May 2018

page(s)

491-499

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16 May 2018

Apoptosis-independent functions of killer caspases.

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The Parkinson disease protein leucine-rich repeat kinase 2 transduces death signals via Fas-associated protein with death domain and caspase-8 in a cellular model of neurodegeneration

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Apoptotic and non-apoptotic roles of caspases in neuronal physiology and pathophysiology

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12 December 2020

Caspase-cleaved amyloid precursor protein in Alzheimer's disease.

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Propofol may protect PC12 cells from β-amyloid₂₅₋₃₅ induced apoptosis through the GSK-3β signaling pathway.

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Intraneuronal Aβ accumulation induces hippocampal neuron hyperexcitability through A-type K(+) current inhibition mediated by activation of caspases and GSK-3.

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Amino-truncated amyloid beta-peptide (Abeta5-40/42) produced from caspase-cleaved amyloid precursor protein is deposited in Alzheimer's disease brain.

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12 December 2020

Parthenolide protects human lens epithelial cells from oxidative stress-induced apoptosis via inhibition of activation of caspase-3 and caspase-9

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12 December 2020

Fas and activated caspase 8 in normal, Alzheimer and multiple infarct brains

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A macromolecular structure favouring microtubule assembly in NGF-differentiated pheochromocytoma cells (PC12).

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Neurotrophic Peptides: Potential Drugs for Treatment of Amyotrophic Lateral Sclerosis and Alzheimer's disease

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beta-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicity

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10.1007/S12031-015-0498-5

Identifiers

DOI

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16 May 2018

1 reference

16 May 2018