Anti-inflammatory cytokines hepatocyte growth factor and interleukin-11 are over-expressed in Polycythemia vera and contribute to the growth of clonal erythroblasts independently of JAK2V617F. (Q53720651)

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English	Anti-inflammatory cytokines hepatocyte growth factor and interleukin-11 are over-expressed in Polycythemia vera and contribute to the growth of clonal erythroblasts independently of JAK2V617F.	scientific article

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scholarly article

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Europe PubMed Central

PubMed publication ID

18 May 2018

http://europepmc.org/abstract/MED/21042281

Anti-inflammatory cytokines hepatocyte growth factor and interleukin-11 are over-expressed in Polycythemia vera and contribute to the growth of clonal erythroblasts independently of JAK2V617F. (English)

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Europe PubMed Central

PubMed publication ID

18 May 2018

http://europepmc.org/abstract/MED/21042281

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polycythemia vera

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based on heuristic

inferred from title

Cédric Cleyrat

2

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Europe PubMed Central

PubMed publication ID

18 May 2018

http://europepmc.org/abstract/MED/21042281

author name string

M Boissinot

1

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Europe PubMed Central

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18 May 2018

http://europepmc.org/abstract/MED/21042281

M Vilaine

3

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Europe PubMed Central

PubMed publication ID

18 May 2018

http://europepmc.org/abstract/MED/21042281

Y Jacques

4

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Europe PubMed Central

PubMed publication ID

18 May 2018

http://europepmc.org/abstract/MED/21042281

I Corre

5

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Europe PubMed Central

PubMed publication ID

18 May 2018

http://europepmc.org/abstract/MED/21042281

S Hermouet

6

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18 May 2018

http://europepmc.org/abstract/MED/21042281

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publication date

1 November 2010

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18 May 2018

http://europepmc.org/abstract/MED/21042281

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Europe PubMed Central

PubMed publication ID

18 May 2018

http://europepmc.org/abstract/MED/21042281

30

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Europe PubMed Central

PubMed publication ID

18 May 2018

http://europepmc.org/abstract/MED/21042281

8

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Europe PubMed Central

PubMed publication ID

18 May 2018

http://europepmc.org/abstract/MED/21042281

990-1001

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Europe PubMed Central

PubMed publication ID

18 May 2018

http://europepmc.org/abstract/MED/21042281

JAK2 mutation and disease phenotype: a double L611V/V617F in cis mutation of JAK2 is associated with isolated erythrocytosis and increased activation of AKT and ERK1/2 rather than STAT5.

1 reference

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Drug development of MET inhibitors: targeting oncogene addiction and expedience

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BCR-ABL induces opposite phenotypes in murine ES cells according to STAT3 activation levels

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Erythropoietin-independent erythroid colony formation by bone marrow progenitors exposed to interleukin-11 and interleukin-8.

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The hepatocyte growth factor/Met pathway controls proliferation and apoptosis in multiple myeloma.

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Standardization and comparison of endogenous erythroid colony assays performed with bone marrow or blood progenitors for the diagnosis of polycythemia vera

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Genetic factors in chronic inflammation: single nucleotide polymorphisms in the STAT-JAK pathway, susceptibility to DNA damage and Crohn's disease in a New Zealand population.

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Frequent reduction or absence of detection of the JAK2-mutated clone in JAK2V617F-positive patients within the first years of hydroxyurea therapy

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Abnormal production of interleukin (IL)-11 and IL-8 in polycythaemia vera

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The current status and the future of JAK2 inhibitors for the treatment of myeloproliferative diseases

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Global cytokine analysis in myeloproliferative disorders

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T cells from patients with polycythemia vera elaborate growth factors which contribute to endogenous erythroid and megakaryocyte colony formation

1 reference

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A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia vera

1 reference

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The threshold of gp130-dependent STAT3 signaling is critical for normal regulation of hematopoiesis

1 reference

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Pathologic consequences of STAT3 hyperactivation by IL-6 and IL-11 during hematopoiesis and lymphopoiesis

1 reference

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JAK2 haplotype is a major risk factor for the development of myeloproliferative neoplasms

1 reference

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High molecular response rate of polycythemia vera patients treated with pegylated interferon alpha-2a

1 reference

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A germline JAK2 SNP is associated with predisposition to the development of JAK2(V617F)-positive myeloproliferative neoplasms

1 reference

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Induction of hepatocyte growth factor activator gene expression under hypoxia activates the hepatocyte growth factor/c-Met system via hypoxia inducible factor-1 in pancreatic cancer

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Tissue inhibitor of metalloproteinases-1 promotes liver metastasis by induction of hepatocyte growth factor signaling.

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A gain-of-function mutation of JAK2 in myeloproliferative disorders

1 reference

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Genetic complexity of myeloproliferative neoplasms.

1 reference

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JAK2V617F expression in murine hematopoietic cells leads to MPD mimicking human PV with secondary myelofibrosis.

1 reference

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In essential thrombocythemia, multiple JAK2-V617F clones are present in most mutant-positive patients: a new disease paradigm.

1 reference

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Dual implication of fibrogenic cytokines in the pathogenesis of fibrosis and myeloproliferation in myeloid metaplasia with myelofibrosis

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The JAK2-V617F mutation is frequently present at diagnosis in patients with essential thrombocythemia and polycythemia vera

1 reference

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Concordance of assays designed for the quantification of JAK2V617F: a multicenter study

1 reference

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Hepatocyte growth factor plays roles in the induction and autocrine maintenance of bone marrow stromal cell IL-11, SDF-1 alpha, and stem cell factor.

1 reference

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Soluble angiogenic factors: implications for chronic myeloproliferative disorders

1 reference

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Numerical gain and structural rearrangements of JAK2, identified by FISH, characterize both JAK2617V>F-positive and -negative patients with Ph-negative MPD, myelodysplasia, and B-lymphoid neoplasms

1 reference

https://api.crossref.org/works/10.1038%2FONC.2010.479

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Polycythemia vera is not initiated by JAK2V617F mutation.

1 reference

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A common JAK2 haplotype confers susceptibility to myeloproliferative neoplasms.

1 reference

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Serum interleukin (IL)-1, IL-2, sIL-2Ra, IL-6 and thrombopoietin levels in patients with chronic myeloproliferative diseases

1 reference

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Interferon-alpha activates multiple STAT signals and down-regulates c-Met in primary human hepatocytes

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c-Met and hepatocyte growth factor: potential as novel targets in cancer therapy

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Clonal analysis of deletions on chromosome 20q and JAK2-V617F in MPD suggests that del20q acts independently and is not one of the predisposing mutations for JAK2-V617F.

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RNA interference reveals that ligand-independent met activity is required for tumor cell signaling and survival

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Multiple myeloma cell killing by depletion of the MET receptor tyrosine kinase

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Hepatocyte growth factor signalling stimulates hypoxia inducible factor-1 (HIF-1) activity in HepG2 hepatoma cells

1 reference

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Ratio of mutant JAK2-V617F to wild-type Jak2 determines the MPD phenotypes in transgenic mice

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The World Health Organization (WHO) classification of the myeloid neoplasms

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Therapeutic potential of Janus-activated kinase-2 inhibitors for the management of myelofibrosis

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Expression of Jak2V617F causes a polycythemia vera-like disease with associated myelofibrosis in a murine bone marrow transplant model

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Polycythemia vera megakaryocytes but not megakaryocytes from normal controls and patients with smokers polyglobuly spontaneously express IL-6 and IL-6R and secrete IL-6.

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Abnormal cytokine production by bone marrow stromal cells of multiple myeloma patients in response to RPMI8226 myeloma cells.

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10.1038/ONC.2010.479

1 reference

Europe PubMed Central

PubMed publication ID

18 May 2018

http://europepmc.org/abstract/MED/21042281

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

18 May 2018

http://europepmc.org/abstract/MED/21042281

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