Bcl-2 expression delays mammary tumor development in dimethylbenz(a)anthracene-treated transgenic mice. (Q54004293)

23 May 2018

title

Bcl-2 expression delays mammary tumor development in dimethylbenz(a)anthracene-treated transgenic mice. (English)

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23 May 2018

author name string

Murphy KL

1

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23 May 2018

Kittrell FS

2

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23 May 2018

Gay JP

3

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23 May 2018

Jäger R

4

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23 May 2018

Medina D

5

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23 May 2018

Rosen JM

6

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23 May 2018

language of work or name

English

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publication date

1 November 1999

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23 May 2018

published in

Oncogene

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23 May 2018

volume

18

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23 May 2018

page(s)

6597-6604

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23 May 2018

issue

47

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23 May 2018

https://scigraph.springernature.com/pub.10.1038/sj.onc.1203099

exact match

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cites work

Bcl-2 inhibits p53 nuclear import following DNA damage

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Kinetics of mammary epithelial cell proliferation in pituitary isografted BALB/c mice

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Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation

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Mediation of c-Myc-induced apoptosis by p53.

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Overexpression of Bcl-2 inhibits alveolar cell apoptosis during involution and accelerates c-myc-induced tumorigenesis of the mammary gland in transgenic mice.

21 January 2018

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The proto-oncogene Bcl-2 and its role in regulating apoptosis

21 January 2018

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Delay of dimethylbenz[a]anthracene-induced mammary tumorigenesis in transgenic mice by apoptosis induced by an unusual mutant p53 protein

21 January 2018

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bcl-2 overexpression inhibits cell death and promotes the morphogenesis, but not tumorigenesis of human mammary epithelial cells

21 January 2018

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Mammary tumorigenesis in chemical carcinogen-treated mice. I. Incidence in BALB-c and C57BL mice.

21 January 2018

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The p53-binding protein 53BP2 also interacts with Bc12 and impedes cell cycle progression at G2/M

21 January 2018

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Human keratin-1.bcl-2 transgenic mice aberrantly express keratin 6, exhibit reduced sensitivity to keratinocyte cell death induction, and are susceptible to skin tumor formation.

21 January 2018

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DNA damage can induce apoptosis in proliferating lymphoid cells via p53-independent mechanisms inhibitable by Bcl-2

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Diminished cell proliferation associated with the death-protective activity of Bcl-2

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7 January 2021

Expression of p53 and bcl-2 in correlation to clinicopathological parameters, hormone receptor status and DNA ploidy in breast cancers

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7 January 2021

Pentagon medical school threatened again

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7 January 2021

Estrogen and progesterone receptors, c-erbB-2, p53, and Bcl-2 in thirty-three invasive micropapillary breast carcinomas

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1203099

7 January 2021

The cell death inhibitor Bcl-2 and its homologues influence control of cell cycle entry

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7 January 2021

bcl-2 expression in the spectrum of preinvasive breast lesions

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7 January 2021

Identifiers

DOI

10.1038/SJ.ONC.1203099

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23 May 2018

PubMed ID

10597264

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23 May 2018