c-Fos suppresses systemic inflammatory response to endotoxin (Q57675135)

1

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Masayoshi Kuwahara

2

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Yasunari Takada

3

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Kenta Maruyama

4

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Tomohiro Kawaguchi

5

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Hirokazu Tsubone

6

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Hiromichi Ishikawa

The immunopathogenesis of sepsis

7

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publication date

28 March 2006

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published in

International Immunology

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volume

18

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issue

5

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page(s)

671-677

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cites work

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Fosl1 is a transcriptional target of c-Fos during osteoclast differentiation

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Osteoclasts, mononuclear phagocytes, and c-Fos: new insight into osteoimmunology

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Nrf1 in a complex with fosB, c-jun, junD and ATF2 forms the AP1 component at the TNF alpha promoter in stimulated mast cells

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Cutting edge: different Toll-like receptor agonists instruct dendritic cells to induce distinct Th responses via differential modulation of extracellular signal-regulated kinase-mitogen-activated protein kinase and c-Fos.

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A Toll-like receptor 2 ligand stimulates Th2 responses in vivo, via induction of extracellular signal-regulated kinase mitogen-activated protein kinase and c-Fos in dendritic cells

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Simvastatin augments lipopolysaccharide-induced proinflammatory responses in macrophages by differential regulation of the c-Fos and c-Jun transcription factors

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Dominant-negative effect of the c-fos family gene products on inducible NO synthase expression in macrophages

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Genetic Deletion of the Tumor Necrosis Factor Receptor p60 or p80 Sensitizes Macrophages to Lipopolysaccharide-induced Nuclear Factor-κB, Mitogen-activated Protein Kinases, and Apoptosis

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A human homologue of the Drosophila Toll protein signals activation of adaptive immunity

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A protein C deficiency exacerbates inflammatory and hypotensive responses in mice during polymicrobial sepsis in a cecal ligation and puncture model

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Targeted disruption of pyrin, the FMF protein, causes heightened sensitivity to endotoxin and a defect in macrophage apoptosis

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TRAIL-R as a negative regulator of innate immune cell responses.

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Dok-1 and Dok-2 are negative regulators of lipopolysaccharide-induced signaling

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The ubiquitin-modifying enzyme A20 is required for termination of Toll-like receptor responses

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Downregulation of lipopolysaccharide response in Drosophila by negative crosstalk between the AP1 and NF-kappaB signaling modules.

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c-Fos: a key regulator of osteoclast-macrophage lineage determination and bone remodeling

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