High Mda-7 expression promotes malignant cell survival and p38 MAP kinase activation in chronic lymphocytic leukemia (Q58041746)

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H Gary-Gouy

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A Portier

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F Davi

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P Galanaud

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A Dalloul

https://scigraph.springernature.com/pub.10.1038/sj.leu.2404073

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publication date

12 January 2006

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published in

Leukemia

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volume

20

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issue

3

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page(s)

498-504

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exact match

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cites work

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The protein product of the tumor suppressor gene, melanoma differentiation-associated gene 7, exhibits immunostimulatory activity and is designated IL-24.

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mda-7 (IL-24) Mediates selective apoptosis in human melanoma cells by inducing the coordinated overexpression of the GADD family of genes by means of p38 MAPK.

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A MAP kinase targeted by endotoxin and hyperosmolarity in mammalian cells

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Independent regulation of JNK/p38 mitogen-activated protein kinases by metabolic oxidative stress in the liver

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Osmotic shock induces G1 arrest through p53 phosphorylation at Ser33 by activated p38MAPK without phosphorylation at Ser15 and Ser20.

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A selective defect in IgM antigen receptor synthesis and transport causes loss of cell surface IgM expression on tolerant B lymphocytes.

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Human CD5 promotes B-cell survival through stimulation of autocrine IL-10 production

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CD5-mediated negative regulation of antigen receptor-induced growth signals in B-1 B cells

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Theophylline-induced B-CLL apoptosis is partly dependent on cyclic AMP production but independent of CD38 expression and endogenous IL-10 production

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Constitutive activation of the MAPkinase p38 is critical for MMP-9 production and survival of B-CLL cells on bone marrow stromal cells

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CD5-induced apoptosis of B cells in some patients with chronic lymphocytic leukemia

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7 January 2021