Adipokines and Adipocyte Function in Clock Mutant Mice That Retain Melatonin Rhythmicity (Q59709675)

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12 February 2020

title

Adipokines and adipocyte function in Clock mutant mice that retain melatonin rhythmicity (English)

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12 February 2020

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2

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12 February 2020

author name string

Athena Voultsios

series ordinal

3

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12 February 2020

Nicole Wight

series ordinal

4

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David J Kennaway

series ordinal

1

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12 February 2020

publication date

15 September 2011

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12 February 2020

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volume

20

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issue

2

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page(s)

295-305

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Health disorders of shift workers

12 February 2020

cites work

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Disruption of the clock components CLOCK and BMAL1 leads to hypoinsulinaemia and diabetes

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Circadian clocks are resounding in peripheral tissues

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BMAL1 and CLOCK, two essential components of the circadian clock, are involved in glucose homeostasis

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Functional central rhythmicity and light entrainment, but not liver and muscle rhythmicity, are Clock independent.

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Transcriptional profiling in the adrenal gland reveals circadian regulation of hormone biosynthesis genes and nucleosome assembly genes

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Characterization of Peripheral Circadian Clocks in Adipose Tissues

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Food entrainment of circadian gene expression altered in PPARalpha-/- brown fat and heart

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Identification of Rev-erbalpha as a physiological repressor of apoC-III gene transcription

31 March 2020

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Circadian clock feedback cycle through NAMPT-mediated NAD+ biosynthesis

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Functional expression of MT2 (Mel1b) melatonin receptors in human PAZ6 adipocytes

31 March 2020

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inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1038/OBY.2011.276

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:21918578%20AND%20SRC:MED&resulttype=core&format=json

12 February 2020

1 reference