β-Amyloid-induced calcium influx induces apoptosis in culture by oxidative stress rather than tau phosphorylation (Q60530540)

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5 November 2019

title

Beta-amyloid-induced calcium influx induces apoptosis in culture by oxidative stress rather than tau phosphorylation (English)

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5 November 2019

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F J Ekinci

1

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5 November 2019

M D Linsley

series ordinal

2

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5 November 2019

T B Shea

series ordinal

3

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5 November 2019

publication date

1 March 2000

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5 November 2019

published in

Molecular Brain Research

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5 November 2019

volume

76

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5 November 2019

issue

2

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5 November 2019

page(s)

389-395

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Apoptosis of retrogradely degenerating neurons occurs in association with the accumulation of perikaryal mitochondria and oxidative damage to the nucleus

5 November 2019

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Abnormal phosphorylation of tau and the mechanism of Alzheimer neurofibrillary degeneration: sequestration of microtubule-associated proteins 1 and 2 and the disassembly of microtubules by the abnormal tau

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beta-Amyloid peptide fragment 25-35 potentiates the calcium-dependent release of excitatory amino acids from depolarized hippocampal slices.

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Accumulation of abnormally phosphorylated tau precedes the formation of neurofibrillary tangles in Alzheimer's disease

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Hydrogen peroxide mediates amyloid beta protein toxicity

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Do defects in mitochondrial energy metabolism underlie the pathology of neurodegenerative diseases?

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beta-amyloid fibrils induce tau phosphorylation and loss of microtubule binding.

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Hyperphosphorylation of Tau and filopodial retraction following microinjection of protein kinase C catalytic subunits

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beta-Amyloid neurotoxicity: a discussion of in vitro findings

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Activation of the L voltage-sensitive calcium channel by mitogen-activated protein (MAP) kinase following exposure of neuronal cells to beta-amyloid. MAP kinase mediates beta-amyloid-induced neurodegeneration.

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Apoptosis mediated neurotoxicity induced by chronic application of beta amyloid fragment 25-35.

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Catecholamines potentiate amyloid beta-peptide neurotoxicity: involvement of oxidative stress, mitochondrial dysfunction, and perturbed calcium homeostasis.

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Alzheimer's presenilin mutation sensitizes neural cells to apoptosis induced by trophic factor withdrawal and amyloid beta-peptide: involvement of calcium and oxyradicals.

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Neurodegeneration mediated by glutamate and beta-amyloid peptide: a comparison and possible interaction

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beta-Amyloid protein amplifies calcium signalling in central neurons from the adult mouse.

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beta-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicity

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Defective brain microtubule assembly in Alzheimer's disease

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Calpain-mediated proteolysis of microtubule-associated protein 2 (MAP-2) is inhibited by phosphorylation by cAMP-dependent protein kinase, but not by Ca2+/calmodulin-dependent protein kinase II.

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Beta-amyloid protein increases the vulnerability of cultured cortical neurons to excitotoxic damage

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Calcium influx and the Ca2+-calmodulin complex are involved in interferon-gamma-induced expression of HLA class II molecules on HL-60 cells

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Beta/A4-evoked degeneration of differentiated SH-SY5Y human neuroblastoma cells

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Phosphorylation, calpain proteolysis and tubulin binding of recombinant human tau isoforms.

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7 January 2021

Tau in Alzheimer's disease.

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Calcium-destabilizing and neurodegenerative effects of aggregated beta-amyloid peptide are attenuated by basic FGF.

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7 January 2021

Neurons may live for decades with neurofibrillary tangles.

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Calcium/calmodulin-dependent protein kinase IIalpha mediates activation of mitogen-activated protein kinase and cytosolic phospholipase A2 in norepinephrine-induced arachidonic acid release in rabbit aortic smooth muscle cells

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7 January 2021

RNA oxidation is a prominent feature of vulnerable neurons in Alzheimer's disease.

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7 January 2021

3-Hydroxykynurenine, an endogenous oxidative stress generator, causes neuronal cell death with apoptotic features and region selectivity.

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7 January 2021

Binding of amyloid beta-peptide to mitochondrial hydroxyacyl-CoA dehydrogenase (ERAB): regulation of an SDR enzyme activity with implications for apoptosis in Alzheimer's disease

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7 January 2021

Differences between vascular and plaque core amyloid in Alzheimer's disease

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7 January 2021

Apoptotic death in an in vitro model of neuronal oxidative stress.

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7 January 2021

Increased surface phosphatidylserine is an early marker of neuronal apoptosis

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7 January 2021

Voltage-dependent potentiation of L-type Ca2+ channels due to phosphorylation by cAMP-dependent protein kinase

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7 January 2021

Isolation and quantification of soluble Alzheimer's beta-peptide from biological fluids

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7 January 2021

Calcium influx recruits an additional class of kinases to hyperphosphorylate tau

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7 January 2021

Calcium influx into human neuroblastoma cells induces ALZ-50 immunoreactivity: involvement of calpain-mediated hydrolysis of protein kinase C

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7 January 2021

Beta-amyloid and ionophore A23187 evoke tau hyperphosphorylation by distinct intracellular pathways: differential involvement of the calpain/protein kinase C system

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7 January 2021

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Amyloid-β Deposition in Alzheimer Transgenic Mice Is Associated with Oxidative Stress

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7 January 2021

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7 January 2021

Neurotoxicity of A beta amyloid protein in vitro is not altered by calcium channel blockade

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7 January 2021

The endoplasmic reticulum stress-responsive protein GRP78 protects neurons against excitotoxicity and apoptosis: suppression of oxidative stress and stabilization of calcium homeostasis.

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7 January 2021

Hyperphosphorylated tau in SY5Y cells: similarities and dissimilarities to abnormally hyperphosphorylated tau from Alzheimer disease brain

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7 January 2021

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Identifiers

DOI

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10762716%20AND%20SRC:MED&resulttype=core&format=json

5 November 2019

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