Oncogenes and tumor angiogenesis: the HPV-16 E6 oncoprotein activates the vascular endothelial growth factor (VEGF) gene promoter in a p53 independent manner (Q62566479)

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English	Oncogenes and tumor angiogenesis: the HPV-16 E6 oncoprotein activates the vascular endothelial growth factor (VEGF) gene promoter in a p53 independent manner	article

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1 November 2019

Oncogenes and tumor angiogenesis: the HPV-16 E6 oncoprotein activates the vascular endothelial growth factor (VEGF) gene promoter in a p53 independent manner (English)

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1 November 2019

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tumor angiogenesis

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López-Ocejo O

1

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1 November 2019

Viloria-Petit A

2

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1 November 2019

Bequet-Romero M

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Mukhopadhyay D

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Rak J

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Kerbel RS

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1 September 2000

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4611-4620

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1 November 2019

https://scigraph.springernature.com/pub.10.1038/sj.onc.1203817

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Identification of the HPV-16 E6 protein from transformed mouse cells and human cervical carcinoma cell lines.

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How tumors become angiogenic

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Normal keratinization in a spontaneously immortalized aneuploid human keratinocyte cell line

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Essential role for oncogenic Ras in tumour maintenance

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Integration of human papillomavirus type 16 into cellular DNA of cervical carcinoma: preferential deletion of the E2 gene and invariable retention of the long control region and the E6/E7 open reading frames

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Degradation of p53 can be targeted by HPV E6 sequences distinct from those required for p53 binding and trans-activation

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The angiopoietins: Yin and Yang in angiogenesis

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Transcriptional activation of several heterologous promoters by the E6 protein of human papillomavirus type 16

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Overexpression of vascular permeability factor/vascular endothelial growth factor and its receptors in psoriasis

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HPV16 E6 oncoprotein stimulates the transforming growth factor-beta 1 promoter in fibroblasts through a specific GC-rich sequence

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The human papilloma virus-16 E7 oncoprotein is able to bind to the retinoblastoma gene product

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Down-regulation of vascular endothelial growth factor in a human colon carcinoma cell line transfected with an antisense expression vector specific for c-src

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The role of vascular endothelial growth factor in pathological angiogenesis

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Heterozygous embryonic lethality induced by targeted inactivation of the VEGF gene

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Transforming growth factor-alpha-induced transcriptional activation of the vascular permeability factor (VPF/VEGF) gene requires AP-2-dependent DNA binding and transactivation.

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Post-transcriptional regulation of vascular endothelial growth factor mRNA by the product of the VHL tumor suppressor gene

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Epidermal growth factor stimulates vascular endothelial growth factor production by human malignant glioma cells: a model of glioblastoma multiforme pathophysiology

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Vascular permeability factor (vascular endothelial growth factor) expression and angiogenesis in cervical neoplasia

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Signaling vascular morphogenesis and maintenance

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Patterns and emerging mechanisms of the angiogenic switch during tumorigenesis

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Do HPV-negative cervical carcinomas exist??revisited

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Expression of epidermal growth factor receptor and human papillomavirus E6/E7 proteins in cervical carcinoma cells

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Transactivation of prothymosin alpha and c-myc promoters by human papillomavirus type 16 E6 protein

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VEGF/VPF: the angiogenesis factor found?

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The molecular genetics of cervical carcinoma

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Hypoxia-inducible protein binding to vascular endothelial growth factor mRNA and its modulation by the von Hippel-Lindau protein

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Humanization of a mouse monoclonal antibody that blocks the epidermal growth factor receptor: recovery of antagonistic activity

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The von Hippel-Lindau tumor suppressor gene product interacts with Sp1 to repress vascular endothelial growth factor promoter activity

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Hypoxic induction of human vascular endothelial growth factor expression through c-Src activation

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Oncogenes as inducers of tumor angiogenesis

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Viral transcription in human keratinocyte cell lines immortalized by human papillomavirus type-16.

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The HPV-16 E6 and E6-AP complex functions as a ubiquitin-protein ligase in the ubiquitination of p53

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The significance of atypical vessels and neovascularization in cervical neoplasia

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Human papillomavirus type 16 E6 and E7 oncogenes abrogate radiation-induced DNA damage responses in vivo through p53-dependent and p53-independent pathways

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Different angiogenic pathways in human cervical cancers

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Sequential development of an angiogenic phenotype by human fibroblasts progressing to tumorigenicity

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Human papillomavirus is a necessary cause of invasive cervical cancer worldwide

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1 reference

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