HIV-1 Vpr induces ATM-dependent cellular signal with enhanced homologous recombination (Q64388284)

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30 December 2019

title

HIV-1 Vpr induces ATM-dependent cellular signal with enhanced homologous recombination (English)

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30 December 2019

5

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30 December 2019

Hitoshi Kurumizaka

10

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30 December 2019

author name string

Nakai-Murakami C

1

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30 December 2019

Shimura M

2

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30 December 2019

Kinomoto M

3

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30 December 2019

Takizawa Y

4

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30 December 2019

Taguchi T

6

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30 December 2019

Hoshino S

7

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30 December 2019

Miyagawa K

8

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30 December 2019

Sata T

9

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30 December 2019

Yuo A

11

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30 December 2019

Ishizaka Y

12

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30 December 2019

language of work or name

English

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publication date

18 September 2006

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30 December 2019

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30 December 2019

volume

26

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30 December 2019

issue

4

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30 December 2019

page(s)

477-486

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1209831

30 December 2019

exact match

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HIV-1 VPR modulates cell cycle G2 M transition through an alternative cellular mechanism other than the classic mitotic checkpoints

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HIV-1 Vpr increases viral expression by manipulation of the cell cycle: a mechanism for selection of Vpr in vivo

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Cell cycle arrest in G2/M promotes early steps of infection by human immunodeficiency virus

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Human immunodeficiency virus type 1 viral protein R (Vpr) arrests cells in the G2 phase of the cell cycle by inhibiting p34cdc2 activity

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Functional and structural characterization of synthetic HIV-1 Vpr that transduces cells, localizes to the nucleus, and induces G2 cell cycle arrest

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Characterization of HIV-1 vpr nuclear import: analysis of signals and pathways

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Homologous pairing promoted by the human Rad52 protein.

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7 January 2021

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7 January 2021

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7 January 2021

BRCA1 physically associates with p53 and stimulates its transcriptional activity.

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Human immunodeficiency virus type 1 Vpr-mediated G2 arrest requires Rad17 and Hus1 and induces nuclear BRCA1 and gamma-H2AX focus formation.

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7 January 2021