A senescence program controlled by p53 and p16INK4a contributes to the outcome of cancer therapy (Q74117541)

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9 November 2019

title

A senescence program controlled by p53 and p16INK4a contributes to the outcome of cancer therapy (English)

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9 November 2019

4

Soyoung Lee

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9 November 2019

author name string

Clemens A Schmitt

1

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9 November 2019

Jordan S Fridman

2

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9 November 2019

Meng Yang

3

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9 November 2019

Eugene Baranov

5

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9 November 2019

Robert M Hoffman

6

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9 November 2019

Scott W Lowe

7

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9 November 2019

language of work or name

English

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publication date

1 May 2002

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9 November 2019

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9 November 2019

volume

109

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9 November 2019

page(s)

335-346

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9 November 2019

issue

3

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The c-myc oncogene driven by immunoglobulin enhancers induces lymphoid malignancy in transgenic mice

9 November 2019

cites work

1 reference

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7 January 2021

Involvement of the cyclin-dependent kinase inhibitor p16 (INK4a) in replicative senescence of normal human fibroblasts

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7 January 2021

Dual inactivation of RB and p53 pathways in RAS-induced melanomas

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7 January 2021

Genetic determinants of response to chemotherapy in transgenic mouse mammary and salivary tumors

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7 January 2021

Requirement for p53 and p21 to sustain G2 arrest after DNA damage

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7 January 2021

Disruption of p53 in human cancer cells alters the responses to therapeutic agents

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7 January 2021

p16INK4A and p19ARF act in overlapping pathways in cellular immortalization

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7 January 2021

E1A signaling to p53 involves the p19(ARF) tumor suppressor

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7 January 2021

DNA damage triggers a prolonged p53-dependent G1 arrest and long-term induction of Cip1 in normal human fibroblasts

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7 January 2021

A biomarker that identifies senescent human cells in culture and in aging skin in vivo

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7 January 2021

Disruption of the ARF-Mdm2-p53 tumor suppressor pathway in Myc-induced lymphomagenesis

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7 January 2021

Regulation of p16CDKN2 expression and its implications for cell immortalization and senescence

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7 January 2021

The serial cultivation of human diploid cell strains

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7 January 2021

Apoptosis: a link between cancer genetics and chemotherapy

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7 January 2021

Loss of p16Ink4a confers susceptibility to metastatic melanoma in mice

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7 January 2021

p53-dependent apoptosis modulates the cytotoxicity of anticancer agents

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7 January 2021

Differential effects of p19(Arf) and p16(Ink4a) loss on senescence of murine bone marrow-derived preB cells and macrophages

1 reference

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7 January 2021

Agents that cause DNA double strand breaks lead to p16INK4a enrichment and the premature senescence of normal fibroblasts

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7 January 2021

Bcl-2 mediates chemoresistance in matched pairs of primary E(mu)-myc lymphomas in vivo

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7 January 2021

INK4a/ARF mutations accelerate lymphomagenesis and promote chemoresistance by disabling p53.

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7 January 2021

DNA damage responses and chemosensitivity in the E mu-myc mouse lymphoma model.

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7 January 2021

Genetic analysis of chemoresistance in primary murine lymphomas.

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7 January 2021

Dissecting p53 tumor suppressor functions in vivo

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7 January 2021

Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

1 reference

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7 January 2021

The INK4a/ARF network in tumour suppression

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7 January 2021

Parsing Ink4a/Arf: "pure" p16-null mice

1 reference

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7 January 2021

Cellular senescence: mitotic clock or culture shock?

1 reference

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7 January 2021

Radiation-induced senescence-like growth arrest requires TP53 function but not telomere shortening

1 reference

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7 January 2021

Surfing the p53 network

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7 January 2021

Cell-cycle arrest versus cell death in cancer therapy

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7 January 2021

Whole-body optical imaging of green fluorescent protein-expressing tumors and metastases

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7 January 2021

10.1016/S0092-8674(02)00734-1

Identifiers

DOI

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12015983%20AND%20SRC:MED&resulttype=core&format=json

9 November 2019

1 reference