An essential role for REV3 in mammalian cell survival: absence of REV3 induces p53-independent embryonic death (Q74250699)

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9 November 2019

title

An essential role for REV3 in mammalian cell survival: absence of REV3 induces p53-independent embryonic death (English)

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9 November 2019

6

Haruhiko Koseki

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9 November 2019

author name string

Jiyang O-Wang

1

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9 November 2019

Kagemasa Kajiwara

2

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9 November 2019

Kiyoko Kawamura

3

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9 November 2019

Minoru Kimura

4

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9 November 2019

Hiro Miyagishima

5

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9 November 2019

Masatoshi Tagawa

7

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9 November 2019

publication date

1 May 2002

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Biochemical and Biophysical Research Communications

9 November 2019

published in

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9 November 2019

volume

293

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9 November 2019

issue

3

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9 November 2019

page(s)

1132-1137

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Quality control by DNA repair

9 November 2019

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REV3, a Saccharomyces cerevisiae gene whose function is required for induced mutagenesis, is predicted to encode a nonessential DNA polymerase

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A human homolog of the Saccharomyces cerevisiae REV3 gene, which encodes the catalytic subunit of DNA polymerase zeta

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Alternative splicing, genomic structure, and fine chromosome localization of REV3L.

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A full-length cDNA of hREV3 is predicted to encode DNA polymerase zeta for damage-induced mutagenesis in humans

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Molecular cloning, expression and chromosomal localisation of the mouse Rev3l gene, encoding the catalytic subunit of polymerase zeta

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Molecular characterization of seizure-related genes isolated by differential screening

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Disruption of mouse polymerase zeta (Rev3) leads to embryonic lethality and impairs blastocyst development in vitro.

7 January 2021

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Disruption of the developmentally regulated Rev3l gene causes embryonic lethality.

7 January 2021

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Disruption of the Rev3l-encoded catalytic subunit of polymerase zeta in mice results in early embryonic lethality.

7 January 2021

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Sez4 gene encoding an elongation subunit of DNA polymerase zeta is required for normal embryogenesis.

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Interplay of p53 and DNA-repair protein XRCC4 in tumorigenesis, genomic stability and development

7 January 2021

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Late embryonic lethality and impaired V(D)J recombination in mice lacking DNA ligase IV

7 January 2021

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DNA ligase IV deficiency in mice leads to defective neurogenesis and embryonic lethality via the p53 pathway

7 January 2021

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10.1016/S0006-291X(02)00341-8

7 January 2021

Identifiers

DOI

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9 November 2019

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