Endogenous and exogenous ARC in serum withdrawal mediated PC12 cell apoptosis: a new pro-apoptotic role for ARC (Q74460269)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:11536015%20AND%20SRC:MED&resulttype=core&format=json

10 November 2019

title

Endogenous and exogenous ARC in serum withdrawal mediated PC12 cell apoptosis: a new pro-apoptotic role for ARC (English)

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10 November 2019

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Dowds TA

1

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10 November 2019

Sabban EL

series ordinal

2

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10 November 2019

publication date

1 June 2001

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Cell Death & Differentiation

10 November 2019

published in

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10 November 2019

volume

8

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10 November 2019

issue

6

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10 November 2019

page(s)

640-648

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https://scigraph.springernature.com/pub.10.1038/sj.cdd.4400855

10 November 2019

exact match

0 references

cites work

ARC, an inhibitor of apoptosis expressed in skeletal muscle and heart that interacts selectively with caspases

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NMR structure and mutagenesis of the Fas (APO-1/CD95) death domain

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NMR structure and mutagenesis of the FADD (Mort1) death-effector domain

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Solution structure of the RAIDD CARD and model for CARD/CARD interaction in caspase-2 and caspase-9 recruitment

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Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

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RAIDD is a new 'death' adaptor molecule

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Caspases: enemies within

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Inhibition of death receptor signals by cellular FLIP

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Cloning and characterization of cDNAs for novel proteins with glutamic acid-proline dipeptide tandem repeats

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ARC inhibits cytochrome c release from mitochondria and protects against hypoxia-induced apoptosis in heart-derived H9c2 cells

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Death receptors: signaling and modulation

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Caspase-2 (Nedd-2) processing and death of trophic factor-deprived PC12 cells and sympathetic neurons occur independently of caspase-3 (CPP32)-like activity

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7 January 2021

Nedd2 is required for apoptosis after trophic factor withdrawal, but not superoxide dismutase (SOD1) downregulation, in sympathetic neurons and PC12 cells

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Need for caspase-2 in apoptosis of growth-factor-deprived PC12 cells

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Nerve growth factor prevents the death and stimulates the neuronal differentiation of clonal PC12 pheochromocytoma cells in serum-free medium

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A timetable of events during programmed cell death induced by trophic factor withdrawal from neuronal PC12 cells.

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7 January 2021

Multiple agents rescue PC12 cells from serum-free cell death by translation- and transcription-independent mechanisms

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7 January 2021

Nerve growth factor withdrawal-induced cell death in neuronal PC12 cells resembles that in sympathetic neurons

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7 January 2021

Subcellular localization and CARD-dependent oligomerization of the death adaptor RAIDD.

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Caspase-2 is localized at the Golgi complex and cleaves golgin-160 during apoptosis

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Caspases: their intracellular localization and translocation during apoptosis.

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The role of the ubiquitin-proteasome pathway in apoptosis

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Death-effector filaments: novel cytoplasmic structures that recruit caspases and trigger apoptosis

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Caspase-8 is required for cell death induced by expanded polyglutamine repeats.

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E1B 19K inhibits Fas-mediated apoptosis through FADD-dependent sequestration of FLICE.

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Ultrastructural distribution of the death-domain-containing MyD88 protein in HeLa cells

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Aurintricarboxylic acid rescues PC12 cells and sympathetic neurons from cell death caused by nerve growth factor deprivation: correlation with suppression of endonuclease activity

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7 January 2021

The role of estrogens on the proliferation of human breast tumor cells (MCF-7).

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7 January 2021

Cell cycle blockers mimosine, ciclopirox, and deferoxamine prevent the death of PC12 cells and postmitotic sympathetic neurons after removal of trophic support

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Differing temporal roles of Ca2+ and cAMP in nicotine-elicited elevation of tyrosine hydroxylase mRNA.

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7 January 2021

Identifiers

DOI

10.1038/SJ.CDD.4400855

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:11536015%20AND%20SRC:MED&resulttype=core&format=json

10 November 2019

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