TGF-beta induces apoptosis through Smad-mediated expression of DAP-kinase (Q77342591)

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4 December 2019

title

TGF-beta induces apoptosis through Smad-mediated expression of DAP-kinase (English)

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4 December 2019

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Chuan-Wei Jang

1

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4 December 2019

Chun-Han Chen

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4 December 2019

Chun-Chieh Chen

3

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4 December 2019

Jia-yun Chen

4

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4 December 2019

Yi-Hsien Su

5

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4 December 2019

Ruey-Hwa Chen

6

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4 December 2019

publication date

1 January 2002

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4 December 2019

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4 December 2019

volume

4

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4 December 2019

issue

1

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4 December 2019

page(s)

51-58

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https://scigraph.springernature.com/pub.10.1038/ncb731

4 December 2019

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Smad7 is induced by CD40 and protects WEHI 231 B-lymphocytes from transforming growth factor-beta -induced growth inhibition and apoptosis

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Critical role of Smads and AP-1 complex in transforming growth factor-beta -dependent apoptosis

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How cells read TGF-beta signals

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Identification of a novel serine/threonine kinase and a novel 15-kD protein as potential mediators of the gamma interferon-induced cell death

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DAP-kinase is a Ca2+/calmodulin-dependent, cytoskeletal-associated protein kinase, with cell death-inducing functions that depend on its catalytic activity

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DAP kinase activates a p19ARF/p53-mediated apoptotic checkpoint to suppress oncogenic transformation

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DAP-kinase loss of expression in various carcinoma and B-cell lymphoma cell lines: possible implications for role as tumor suppressor gene

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Transcriptional regulation of the transforming growth factor-beta -inducible mouse germ line Ig alpha constant region gene by functional cooperation of Smad, CREB, and AML family members.

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Apoptotic molecular machinery: vastly increased complexity in vertebrates revealed by genome comparisons

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7 January 2021

Profiling expression patterns and isolating differentially expressed genes by cDNA microarray system with colorimetry detection.

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Transforming growth factor-beta (TGF-beta)-induced down-regulation of cyclin A expression requires a functional TGF-beta receptor complex. Characterization of chimeric and truncated type I and type II receptors

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A simplified system for generating recombinant adenoviruses

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Suppression of transforming growth factor-beta-induced apoptosis through a phosphatidylinositol 3-kinase/Akt-dependent pathway

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Ligand-independent activation of transforming growth factor (TGF) beta signaling pathways by heteromeric cytoplasmic domains of TGF-beta receptors

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