Expression of the low affinity neurotrophin receptor p75 in spinal motoneurons in a transgenic mouse model for amyotrophic lateral sclerosis (Q78944558)

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scientific article published on 01 January 2003

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English	Expression of the low affinity neurotrophin receptor p75 in spinal motoneurons in a transgenic mouse model for amyotrophic lateral sclerosis	scientific article published on 01 January 2003

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scholarly article

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18 December 2019

Expression of the low affinity neurotrophin receptor p75 in spinal motoneurons in a transgenic mouse model for amyotrophic lateral sclerosis (English)

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18 December 2019

amyotrophic lateral sclerosis

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J C V M Copray

1

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18 December 2019

D Jaarsma

2

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18 December 2019

B M Küst

3

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18 December 2019

R W G Bruggeman

4

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18 December 2019

I Mantingh

5

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18 December 2019

N Brouwer

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18 December 2019

H W G M Boddeke

7

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18 December 2019

publication date

1 January 2003

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18 December 2019

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18 December 2019

116

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18 December 2019

3

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18 December 2019

685-694

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18 December 2019

The Trk family of neurotrophin receptors

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The p75 neurotrophin receptor and neuronal apoptosis

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ALS-linked SOD1 mutant G85R mediates damage to astrocytes and promotes rapidly progressive disease with SOD1-containing inclusions.

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Commitment to cell death measured by loss of clonogenicity is separable from the appearance of apoptotic markers

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Neurotrophins live or let die: does p75NTR decide?

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Neurotrophins in cell survival/death decisions.

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p75 neurotrophin receptor as a modulator of survival and death decisions

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Neurotrophins: the biological paradox of survival factors eliciting apoptosis

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Neurotrophin receptors: mediators of life and death

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Age-dependent penetrance of disease in a transgenic mouse model of familial amyotrophic lateral sclerosis

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Selective expression of neurotrophin-3 messenger RNA in muscle spindles of the rat

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Neuropathological changes in two lines of mice carrying a transgene for mutant human Cu,Zn SOD, and in mice overexpressing wild type human SOD: a model of familial amyotrophic lateral sclerosis (FALS).

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A low expressor line of transgenic mice carrying a mutant human Cu,Zn superoxide dismutase (SOD1) gene develops pathological changes that most closely resemble those in human amyotrophic lateral sclerosis

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Coupling of the p75 neurotrophin receptor to sphingolipid signaling.

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p75 neurotrophin receptor signaling: mechanisms for neurotrophic modulation of cell stress?

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Expression of nerve growth factor receptor mRNA is developmentally regulated and increased after axotomy in rat spinal cord motoneurons

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Improved survival of injured sciatic nerve Schwann cells in mice lacking the p75 receptor

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Effects of facial nerve injury on mouse motoneurons lacking the p75 low-affinity neurotrophin receptor

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Inhibition of ICE slows ALS in mice

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Benefit of vitamin E, riluzole, and gabapentin in a transgenic model of familial amyotrophic lateral sclerosis

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Motor neuron degeneration in mice that express a human Cu,Zn superoxide dismutase mutation

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Relationship of microglial and astrocytic activation to disease onset and progression in a transgenic model of familial ALS.

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Changes of nerve growth factor synthesis in nonneuronal cells in response to sciatic nerve transection

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The antioxidant N-acetylcysteine does not delay disease onset and death in a transgenic mouse model of amyotrophic lateral sclerosis

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Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mut

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Induction of c-Jun immunoreactivity in spinal cord and brainstem neurons in a transgenic mouse model for amyotrophic lateral sclerosis

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CuZn superoxide dismutase (SOD1) accumulates in vacuolated mitochondria in transgenic mice expressing amyotrophic lateral sclerosis-linked SOD1 mutations

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Nerve growth factor receptor immunostaining in the spinal cord and peripheral nerves in amyotrophic lateral sclerosis

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Motor neurons in Onuf's nucleus and its rat homologues express the p75 nerve growth factor receptor: sexual dimorphism and regulation by axotomy

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Massive mitochondrial degeneration in motor neurons triggers the onset of amyotrophic lateral sclerosis in mice expressing a mutant SOD1

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Bcl-2: prolonging life in a transgenic mouse model of familial amyotrophic lateral sclerosis

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Neuronal death in amyotrophic lateral sclerosis is apoptosis: possible contribution of a programmed cell death mechanism

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Asynchrony and commitment to die during apoptosis

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Caspase-1 and -3 are sequentially activated in motor neuron death in Cu,Zn superoxide dismutase-mediated familial amyotrophic lateral sclerosis

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Identifiers

10.1016/S0306-4522(02)00755-8

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12573711%20AND%20SRC:MED&resulttype=core&format=json

18 December 2019

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12573711%20AND%20SRC:MED&resulttype=core&format=json

18 December 2019

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