Hypoxia-inducible factor-1alpha expression requires PI 3-kinase activity and correlates with Akt1 phosphorylation in invasive breast carcinomas (Q83305663)

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scientific article published on 08 May 2006

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English	Hypoxia-inducible factor-1alpha expression requires PI 3-kinase activity and correlates with Akt1 phosphorylation in invasive breast carcinomas	scientific article published on 08 May 2006

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20 January 2020

Hypoxia-inducible factor-1alpha expression requires PI 3-kinase activity and correlates with Akt1 phosphorylation in invasive breast carcinomas (English)

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20 January 2020

Paul J van Diest

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van Diest PJ

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20 January 2020

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Gort EH

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20 January 2020

Groot AJ

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20 January 2020

Derks van de Ven TL

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20 January 2020

van der Groep P

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20 January 2020

Verlaan I

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20 January 2020

van Laar T

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20 January 2020

van der Wall E

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20 January 2020

Shvarts A

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8 May 2006

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20 January 2020

6123-6127

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20 January 2020

https://scigraph.springernature.com/pub.10.1038/sj.onc.1209643

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Lack of evidence for the involvement of the phosphoinositide 3-kinase/Akt pathway in the activation of hypoxia-inducible factors by low oxygen tension.

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Phosphatidylinositol 3-kinase/Akt signaling is neither required for hypoxic stabilization of HIF-1 alpha nor sufficient for HIF-1-dependent target gene transcription

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AKT activation predicts outcome in breast cancer patients treated with tamoxifen

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Regulation of hypoxia-inducible factor-1alpha protein level during hypoxic conditions by the phosphatidylinositol 3-kinase/Akt/glycogen synthase kinase 3beta pathway in HepG2 cells

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Vascular endothelial growth factor transcriptional activation is mediated by hypoxia-inducible factor 1alpha, HDM2, and p70S6K1 in response to phosphatidylinositol 3-kinase/AKT signaling

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No consent should be needed for using leftover body material for scientific purposes. For.

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