HMGB1 in ischemic and non-ischemic liver after selective warm ischemia/reperfusion in rat (Q83695948)

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26 January 2020

title

HMGB1 in ischemic and non-ischemic liver after selective warm ischemia/reperfusion in rat (English)

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3

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7

Uta Dahmen

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26 January 2020

author name string

Anding Liu

1

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26 January 2020

Olaf Dirsch

2

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Jian Sun

4

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26 January 2020

Hao Jin

5

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26 January 2020

Wei Dong

6

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publication date

24 March 2011

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Histochemistry and Cell Biology

26 January 2020

published in

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volume

135

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issue

5

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26 January 2020

page(s)

443-452

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https://scigraph.springernature.com/pub.10.1007/s00418-011-0802-6

26 January 2020

exact match

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cites work

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Monocytic cells hyperacetylate chromatin protein HMGB1 to redirect it towards secretion

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New EMBO members' review: the double life of HMGB1 chromatin protein: architectural factor and extracellular signal

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High mobility group 1 protein (HMG-1) stimulates proinflammatory cytokine synthesis in human monocytes

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Mechanism of hepatocyte death after ischemia: apoptosis versus necrosis

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High-mobility group box 1 (HMGB1) protein at the crossroads between innate and adaptive immunity

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HMGB1 is an endogenous immune adjuvant released by necrotic cells

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Role of tumor necrosis factor-alpha in the pathophysiologic alterations after hepatic ischemia/reperfusion injury in the rat.

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The role of cytokine networks in the local liver injury following hepatic ischemia/reperfusion in the rat

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How dying cells alert the immune system to danger

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Blockade of receptor for advanced glycation end product (RAGE) attenuates ischemia and reperfusion injury to the liver in mice

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Release of danger signals during ischemic storage of the liver: a potential marker of organ damage?

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Hepatic ischemia/reperfusion injury--a fresh look

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High-mobility group box 1 protein (HMGB1): nuclear weapon in the immune arsenal

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HMGB1: guiding immunity from within

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Antioxidant defense in hepatic ischemia-reperfusion injury is regulated by damage-associated molecular pattern signal molecules

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Post-translational methylation of high mobility group box 1 (HMGB1) causes its cytoplasmic localization in neutrophils.

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The receptor for advanced glycation end products (RAGE) is a cellular binding site for amphoterin. Mediation of neurite outgrowth and co-expression of rage and amphoterin in the developing nervous system

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Activation of Human Umbilical Vein Endothelial Cells Leads to Relocation and Release of High-Mobility Group Box Chromosomal Protein 1

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High mobility group box 1 protein as a marker of hepatocellular injury in human liver transplantation

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Direct induction of acute lung and myocardial dysfunction by liver ischemia and reperfusion

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Molecular mechanisms of hepatic ischemia-reperfusion injury and preconditioning

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High mobility group box 1 protein interacts with multiple Toll-like receptors

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Recovery of liver perfusion after focal outflow obstruction and liver resection

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HMGB1 release induced by liver ischemia involves Toll-like receptor 4 dependent reactive oxygen species production and calcium-mediated signaling

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12 December 2020

Receptor for advanced glycation end product (RAGE)-dependent modulation of early growth response-1 in hepatic ischemia/reperfusion injury.

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Quantitative evaluation and selection of reference genes in a rat model of extended liver resection

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Release of chromatin protein HMGB1 by necrotic cells triggers inflammation

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HMGB1: endogenous danger signaling

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High-Mobility Group Box-1 in Ischemia-Reperfusion Injury of the Heart

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12 December 2020

Activation of gene expression in human neutrophils by high mobility group box 1 protein

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The Microcirculatory Acinar Concept of Normal and Pathological Hepatic Structure

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A novel role for HMGB1 in TLR9-mediated inflammatory responses to CpG-DNA.

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Factors in the pathophysiology of the liver ischemia-reperfusion injury

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TLR4 activation mediates kidney ischemia/reperfusion injury

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The nuclear factor HMGB1 mediates hepatic injury after murine liver ischemia-reperfusion

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HMG-1 as a late mediator of endotoxin lethality in mice

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Protective effects of early CD4(+) T cell reduction in hepatic ischemia/reperfusion injury

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Nucleocytoplasmic shuttling of HMGB1 is regulated by phosphorylation that redirects it toward secretion

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Liver ischemia and reperfusion induces a systemic inflammatory response through Kupffer cell activation

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Molecular mediators of liver ischemia and reperfusion injury: a brief review

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Induction and subcellular localization of high-mobility group box-1 (HMGB1) in the postischemic rat brain

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12 December 2020

Regulated expression and subcellular localization of HMGB1, a chromatin protein with a cytokine function

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12 December 2020

Remote renal injury following partial hepatic ischemia/reperfusion injury in rats

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12 December 2020

Molecular basis for the redox control of nuclear transport of the structural chromatin protein Hmgb1.

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12 December 2020

DAMPs, PAMPs and alarmins: all we need to know about danger

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12 December 2020

10.1007/S00418-011-0802-6

Identifiers

DOI

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26 January 2020

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