Macrophage migration inhibitory factor is a direct target of HBP1-mediated transcriptional repression that is overexpressed in prostate cancer (Q84039307)

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scientific article published on 12 April 2010

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English	Macrophage migration inhibitory factor is a direct target of HBP1-mediated transcriptional repression that is overexpressed in prostate cancer	scientific article published on 12 April 2010

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30 January 2020

Macrophage migration inhibitory factor is a direct target of HBP1-mediated transcriptional repression that is overexpressed in prostate cancer (English)

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30 January 2020

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Y C Chen

1

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X W Zhang

2

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X H Niu

3

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D Q Xin

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W P Zhao

5

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Y Q Na

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Z B Mao

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12 April 2010

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3067-3078

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https://scigraph.springernature.com/pub.10.1038/onc.2010.97

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The HMG-box transcription factor HBP1 is targeted by the pocket proteins and E1A

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Enhanced expression of macrophage migration inhibitory factor in prostatic adenocarcinoma metastases.

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Inhibition of macrophage migration inhibitory factor decreases proliferation and cytokine expression in bladder cancer cells

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Mechanisms and effectors of MIF-dependent promotion of tumourigenesis

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Alterations of the HBP1 transcriptional repressor are associated with invasive breast cancer

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Macrophage migration inhibitory factor deficiency is associated with altered cell growth and reduced susceptibility to Ras-mediated transformation

1 reference

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Macrophage migration inhibitory factor: roles in regulating tumor cell migration and expression of angiogenic factors in hepatocellular carcinoma

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Wnt signalling in stem cells and cancer

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Negative regulation of the Wnt-beta-catenin pathway by the transcriptional repressor HBP1

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Formation of the androgen receptor transcription complex

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Regulation of differentiation by HBP1, a target of the retinoblastoma protein

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High expression of macrophage migration inhibitory factor in human melanoma cells and its role in tumor cell growth and angiogenesis.

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HBP1: a HMG box transcriptional repressor that is targeted by the retinoblastoma family

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The HBP1 transcriptional repressor and the p38 MAP kinase: unlikely partners in G1 regulation and tumor suppression.

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The HBP1 transcriptional repressor participates in RAS-induced premature senescence

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