TWEAK/Fn14 promotes the proliferation and collagen synthesis of rat cardiac fibroblasts via the NF-кB pathway (Q84067885)

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31 January 2020

title

TWEAK/Fn14 promotes the proliferation and collagen synthesis of rat cardiac fibroblasts via the NF-кB pathway (English)

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31 January 2020

author name string

Hui-Na Chen

1

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31 January 2020

De-Jin Wang

2

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31 January 2020

Man-Yi Ren

3

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31 January 2020

Qi-Lei Wang

4

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31 January 2020

Shu-Jian Sui

5

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31 January 2020

publication date

4 May 2012

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Molecular Biology Reports

31 January 2020

published in

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31 January 2020

volume

39

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31 January 2020

issue

8

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31 January 2020

page(s)

8231-8241

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https://scigraph.springernature.com/pub.10.1007/s11033-012-1671-3

31 January 2020

exact match

0 references

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Interplay of matrix metalloproteinases, tissue inhibitors of metalloproteinases and their regulators in cardiac matrix remodeling

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TWEAK induces NF-kappaB2 p100 processing and long lasting NF-kappaB activation.

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Fibroblast growth factor inducible 14 (Fn14) is required for the expression of myogenic regulatory factors and differentiation of myoblasts into myotubes. Evidence for TWEAK-independent functions of Fn14 during myogenesis

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Tumor necrosis factor-related weak inducer of apoptosis augments matrix metalloproteinase 9 (MMP-9) production in skeletal muscle through the activation of nuclear factor-kappaB-inducing kinase and p38 mitogen-activated protein kinase: a potential r

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The Fn14 cytoplasmic tail binds tumour-necrosis-factor-receptor-associated factors 1, 2, 3 and 5 and mediates nuclear factor-kappaB activation

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Expression and function of fibroblast growth factor-inducible 14 in human corneal myofibroblasts

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Tumour necrosis factor-like weak inducer of apoptosis (TWEAK) and its receptor Fn14 during cardiac remodelling in rats.

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The cardiac fibroblast: therapeutic target in myocardial remodeling and failure

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12 December 2020

TRAF2 and cIAP2 involve in TWEAK-induced MMP-9 production in fibroblast-like synoviocytes

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12 December 2020

Involvement of the NF-kappa B/matrix metalloproteinase pathway in cardiac fibrosis of mice lacking guanylyl cyclase/natriuretic peptide receptor A.

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Polymorphism in gene coding for ACE determines different development of myocardial fibrosis in rats

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TWEAK/Fn14 interaction regulates RANTES production, BMP-2-induced differentiation, and RANKL expression in mouse osteoblastic MC3T3-E1 cells

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Tumor necrosis factor-like weak inducer of apoptosis increases the permeability of the neurovascular unit through nuclear factor-kappa B pathway activation.

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Influence of diabetes mellitus on heart failure risk and outcome

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12 December 2020

FGF-inducible 14-kDa protein (Fn14) is regulated via the RhoA/ROCK kinase pathway in cardiomyocytes and mediates nuclear factor-kappaB activation by TWEAK.

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Signaling to NF-kappaB

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