Compensatory contributions of HEXIM1 and HEXIM2 in maintaining the balance of active and inactive positive transcription elongation factor b complexes for control of transcription (Q24294985)

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English	Compensatory contributions of HEXIM1 and HEXIM2 in maintaining the balance of active and inactive positive transcription elongation factor b complexes for control of transcription	scientific article

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scholarly article

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Compensatory contributions of HEXIM1 and HEXIM2 in maintaining the balance of active and inactive positive transcription elongation factor b complexes for control of transcription (English)

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author name string

Jasper H N Yik

1

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Ruichuan Chen

2

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Andrea C Pezda

3

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Qiang Zhou

4

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language of work or name

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publication date

22 April 2005

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Journal of Biological Chemistry

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280

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16368-76

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16

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Elongation by RNA polymerase II: the short and long of it

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Taking a new TAK on tat transactivation

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P-TEFb, a cyclin-dependent kinase controlling elongation by RNA polymerase II

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Flavopiridol inactivates P-TEFb and blocks most RNA polymerase II transcription in vivo

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CDK-9/cyclin T (P-TEFb) is required in two postinitiation pathways for transcription in the C. elegans embryo

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The 7SK small nuclear RNA inhibits the CDK9/cyclin T1 kinase to control transcription

1 reference

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Inhibition of P-TEFb (CDK9/Cyclin T) kinase and RNA polymerase II transcription by the coordinated actions of HEXIM1 and 7SK snRNA

1 reference

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7SK small nuclear RNA binds to and inhibits the activity of CDK9/cyclin T complexes

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Binding of the 7SK snRNA turns the HEXIM1 protein into a P-TEFb (CDK9/cyclin T) inhibitor

1 reference

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MAQ1 and 7SK RNA interact with CDK9/cyclin T complexes in a transcription-dependent manner

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A human immunodeficiency virus type 1 Tat-like arginine-rich RNA-binding domain is essential for HEXIM1 to inhibit RNA polymerase II transcription through 7SK snRNA-mediated inactivation of P-TEFb

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Activation and function of cyclin T-Cdk9 (positive transcription elongation factor-b) in cardiac muscle-cell hypertrophy.

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Ablation of the CLP-1 gene leads to down-regulation of the HAND1 gene and abnormality of the left ventricle of the heart and fetal death

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Suppression of NF-kappaB-dependent gene expression by a hexamethylene bisacetamide-inducible protein HEXIM1 in human vascular smooth muscle cells

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Advanced mammalian gene transfer: high titre retroviral vectors with multiple drug selection markers and a complementary helper-free packaging cell line

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Accurate transcription initiation by RNA polymerase II in a soluble extract from isolated mammalian nuclei

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Transcription elongation factor P-TEFb mediates Tat activation of HIV-1 transcription at multiple stages

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Phosphorylated positive transcription elongation factor b (P-TEFb) is tagged for inhibition through association with 7SK snRNA.

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Specific interaction of Tat with the human but not rodent P-TEFb complex mediates the species-specific Tat activation of HIV-1 transcription

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Structure, expression, and functional characterization of the mouse CLP-1 gene

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Large-scale analysis of the human and mouse transcriptomes

1 reference

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MicroRNAs: genomics, biogenesis, mechanism, and function

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Novel mechanism and factor for regulation by HIV-1 Tat

1 reference

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7 January 2021

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Identifiers

10.1074/JBC.M500912200

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

OpenCitations bibliographic resource ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

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