TMEPAI, a transmembrane TGF-beta-inducible protein, sequesters Smad proteins from active participation in TGF-beta signaling (Q24298286)

Prostate transmembrane protein, androgen induced 1

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R-SMAD binding

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Protein TMEPAI

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7

Kiyotoshi Satoh

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Susumu Itoh

object named as

Susumu Itoh

2

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Peter Ten Dijke

15

object named as

Peter Ten Dijke

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author name string

Yukihide Watanabe

1

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Toshiyasu Goto

3

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Eriko Ohnishi

4

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Masako Inamitsu

5

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Fumiko Itoh

6

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Eliza Wiercinska

8

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Weiwen Yang

9

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Liang Shi

10

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Aya Tanaka

11

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Naoko Nakano

12

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A Mieke Mommaas

13

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Hiroshi Shibuya

14

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Mitsuyasu Kato

EGF- and cell-cycle-regulated STAG1/PMEPA1/ERG1.2 belongs to a conserved gene family and is overexpressed and amplified in breast and ovarian cancer

16

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language of work or name

English

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publication date

15 January 2010

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published in

Molecular Cell

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volume

37

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page(s)

123-34

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issue

1

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cites work

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Mesoderm induction by BMP-4 and -7 heterodimers

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Induction of inhibitory Smad6 and Smad7 mRNA by TGF-beta family members

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A novel androgen-regulated gene, PMEPA1, located on chromosome 20q13 exhibits high level expression in prostate

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Negative regulation of TGF-beta receptor/Smad signal transduction.

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The L45 loop in type I receptors for TGF-beta family members is a critical determinant in specifying Smad isoform activation

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TGFbeta signaling in growth control, cancer, and heritable disorders

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SARA, a FYVE domain protein that recruits Smad2 to the TGFbeta receptor

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Mechanisms of TGF-beta signaling from cell membrane to the nucleus

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Requirement of Xmsx-1 in the BMP-triggered ventralization of Xenopus embryos.

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A truncated activin receptor inhibits mesoderm induction and formation of axial structures in Xenopus embryos

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Identification of Smad7, a TGFbeta-inducible antagonist of TGF-beta signalling

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TGF-beta signaling in tumor suppression and cancer progression

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Synergy and antagonism between Notch and BMP receptor signaling pathways in endothelial cells.

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Identification of STAG1 as a key mediator of a p53-dependent apoptotic pathway

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Regulation of the TGFbeta signalling pathway by ubiquitin-mediated degradation

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TGF-beta and HGF transmit the signals through JNK-dependent Smad2/3 phosphorylation at the linker regions

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ELAC2, a putative prostate cancer susceptibility gene product, potentiates TGF-beta/Smad-induced growth arrest of prostate cells

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The FYVE domain in Smad anchor for receptor activation (SARA) is sufficient for localization of SARA in early endosomes and regulates TGF-beta/Smad signalling

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Phosphorylation of Ser465 and Ser467 in the C terminus of Smad2 mediates interaction with Smad4 and is required for transforming growth factor-beta signaling

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Identification and functional characterization of a Smad binding element (SBE) in the JunB promoter that acts as a transforming growth factor-beta, activin, and bone morphogenetic protein-inducible enhancer.

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Transforming growth factor beta1 induces nuclear export of inhibitory Smad7

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Filamin associates with Smads and regulates transforming growth factor-beta signaling

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Elucidation of Smad requirement in transforming growth factor-beta type I receptor-induced responses

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Hrs, a mammalian master molecule in vesicular transport and protein sorting, suppresses the degradation of ESCRT proteins signal transducing adaptor molecule 1 and 2

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7 January 2021

Dephosphorylation of the linker regions of Smad1 and Smad2/3 by small C-terminal domain phosphatases has distinct outcomes for bone morphogenetic protein and transforming growth factor-beta pathways.

1 reference

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Inhibition of transforming growth factor-beta1-induced signaling and epithelial-to-mesenchymal transition by the Smad-binding peptide aptamer Trx-SARA

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Different Smad2 partners bind a common hydrophobic pocket in Smad2 via a defined proline-rich motif

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The transcriptional co-activator P/CAF potentiates TGF-beta/Smad signaling

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Smad transcription factors

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Disruption of transforming growth factor-beta signaling in ELF beta-spectrin-deficient mice

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Multiple intestinal neoplasia caused by a mutation in the murine homolog of the APC gene

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A dominant mutation that predisposes to multiple intestinal neoplasia in the mouse

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Structural basis of Smad2 recognition by the Smad anchor for receptor activation

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Vertebrate embryonic induction: mesodermal and neural patterning

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Hgs (Hrs), a FYVE domain protein, is involved in Smad signaling through cooperation with SARA.

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Recognition of phosphorylated-Smad2-containing complexes by a novel Smad interaction motif

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Smad4 dependency defines two classes of transforming growth factor {beta} (TGF-{beta}) target genes and distinguishes TGF-{beta}-induced epithelial-mesenchymal transition from its antiproliferative and migratory responses

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Transforming growth factor beta receptor type II inactivation induces the malignant transformation of intestinal neoplasms initiated by Apc mutation.

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TGF-beta switches from tumor suppressor to prometastatic factor in a model of breast cancer progression.

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Endofin acts as a Smad anchor for receptor activation in BMP signaling

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Identifiers

DOI

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Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

4320825

Fatcat ID

release_bbpty7kkrnephh7n73vfluwt6m

1 reference

Fatcat

https://api.fatcat.wiki/v0/release/bbpty7kkrnephh7n73vfluwt6m

24 November 2022

mapped directly with Wikidata item

OpenCitations bibliographic resource ID

4320825

1 reference