Involvement of TIRAP/MAL in signaling for the activation of interferon regulatory factor 3 by lipopolysaccharide (Q24299651)

Interferon regulatory factor 3

GOA release 2020-03-11

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protein homodimerization activity

GOA release 2020-03-11

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GOA release 2020-03-11

Mitsutoshi Yoneyama

3

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author name string

Noriaki Shinobu

1

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Tomokatsu Iwamura

2

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Kazumi Yamaguchi

4

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Wakako Suhara

5

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Yukiko Fukuhara

6

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Fumio Amano

7

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Takashi Fujita

Toll-like receptors in the induction of the innate immune response

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language of work or name

English

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publication date

24 April 2002

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published in

FEBS Letters

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volume

517

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issue

1-3

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page(s)

251-6

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cites work

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Control of IRF-3 activation by phosphorylation

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Direct triggering of the type I interferon system by virus infection: activation of a transcription factor complex containing IRF-3 and CBP/p300.

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Bacterial induction of beta interferon in mice is a function of the lipopolysaccharide component

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p38-dependent activation of interferon regulatory factor 3 by lipopolysaccharide

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Mal (MyD88-adapter-like) is required for Toll-like receptor-4 signal transduction

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TIRAP: an adapter molecule in the Toll signaling pathway

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Induction of IRF-3/-7 kinase and NF-kappaB in response to double-stranded RNA and virus infection: common and unique pathways

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pEF-BOS, a powerful mammalian expression vector

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Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 Gene

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Identification of distinct signaling pathways leading to the phosphorylation of interferon regulatory factor 3

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JNK2 and IKKbeta are required for activating the innate response to viral infection

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Recognition of double-stranded RNA and activation of NF-kappaB by Toll-like receptor 3

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Lipopolysaccharide stimulates the MyD88-independent pathway and results in activation of IFN-regulatory factor 3 and the expression of a subset of lipopolysaccharide-inducible genes

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Deficient signaling in mice devoid of double-stranded RNA-dependent protein kinase

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