IAP antagonists target cIAP1 to induce TNFalpha-dependent apoptosis (Q24300734)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:18022363%20AND%20SRC:MED&resulttype=core&format=json

26 December 2019

title

IAP antagonists target cIAP1 to induce TNFalpha-dependent apoptosis (English)

1 reference

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Receptor (TNFRSF)-interacting serine-threonine kinase 1

26 December 2019

main subject

apoptotic process

0 references

1 reference

Tumor necrosis factor receptor superfamily, member 1a

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

author

W. Wei-Lynn Wong

2

1 reference

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26 December 2019

Afsar U Ahmed

6

1 reference

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26 December 2019

Afsar U Ahmed

7

1 reference

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26 December 2019

Pascal Schneider

15

1 reference

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26 December 2019

David L Vaux

18

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26 December 2019

John Silke

19

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26 December 2019

author name string

James E Vince

1

1 reference

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26 December 2019

Nufail Khan

3

1 reference

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26 December 2019

Rebecca Feltham

4

1 reference

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26 December 2019

Diep Chau

5

1 reference

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26 December 2019

Christopher A Benetatos

8

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26 December 2019

Srinivas K Chunduru

9

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26 December 2019

Stephen M Condon

10

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26 December 2019

Mark McKinlay

11

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26 December 2019

Robert Brink

12

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26 December 2019

Martin Leverkus

13

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26 December 2019

Vinay Tergaonkar

14

1 reference

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26 December 2019

Bernard A Callus

16

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26 December 2019

Frank Koentgen

17

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26 December 2019

language of work or name

English

0 references

publication date

1 November 2007

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26 December 2019

1 reference

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26 December 2019

volume

131

1 reference

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26 December 2019

page(s)

682-693

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26 December 2019

issue

4

1 reference

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The inhibitors of apoptosis (IAPs) as cancer targets

26 December 2019

cites work

1 reference

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The TNFR2-TRAF signaling complex contains two novel proteins related to baculoviral inhibitor of apoptosis proteins

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The lymphotoxin-beta receptor induces different patterns of gene expression via two NF-kappaB pathways

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TNF-induced recruitment and activation of the IKK complex require Cdc37 and Hsp90

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Small-molecule antagonists of apoptosis suppressor XIAP exhibit broad antitumor activity

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Discovery of potent antagonists of the antiapoptotic protein XIAP for the treatment of cancer

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Structural and biochemical basis of apoptotic activation by Smac/DIABLO

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A conserved XIAP-interaction motif in caspase-9 and Smac/DIABLO regulates caspase activity and apoptosis

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7 January 2021

TNF-RII and c-IAP1 mediate ubiquitination and degradation of TRAF2

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7 January 2021

IAPs are functionally non-equivalent and regulate effector caspases through distinct mechanisms

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7 January 2021

Requirement of Hsp90 activity for IkappaB kinase (IKK) biosynthesis and for constitutive and inducible IKK and NF-kappaB activation.

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Cloning and expression of apoptosis inhibitory protein homologs that function to inhibit apoptosis and/or bind tumor necrosis factor receptor-associated factors

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7 January 2021

The tumor necrosis factor receptor 2 signal transducers TRAF2 and c-IAP1 are components of the tumor necrosis factor receptor 1 signaling complex

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7 January 2021

Unlike Diablo/smac, Grim promotes global ubiquitination and specific degradation of X chromosome-linked inhibitor of apoptosis (XIAP) and neither cause apoptosis

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7 January 2021

Smac/Diablo antagonizes ubiquitin ligase activity of inhibitor of apoptosis proteins

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7 January 2021

Smac/DIABLO selectively reduces the levels of c-IAP1 and c-IAP2 but not that of XIAP and livin in HeLa cells

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7 January 2021

Distinct BIR domains of cIAP1 mediate binding to and ubiquitination of tumor necrosis factor receptor-associated factor 2 and second mitochondrial activator of caspases

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7 January 2021

The human anti-apoptotic proteins cIAP1 and cIAP2 bind but do not inhibit caspases

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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Identifiers

DOI

2 references

Consolidated OpenCitations Corpus – April 2017

1320516

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26 December 2019

1320516

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Consolidated OpenCitations Corpus – April 2017

Consolidated OpenCitations Corpus – April 2017

2 references

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