Che-1 activates XIAP expression in response to DNA damage (Q24301894)

X-linked inhibitor of apoptosis

GOA release 2020-03-11

1 reference

positive regulation of transcription by RNA polymerase II

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

C Leonetti

8

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author name string

T Bruno

1

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S Iezzi

2

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F De Nicola

3

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M Di Padova

4

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A Desantis

5

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M Scarsella

6

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M G Di Certo

7

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A Floridi

9

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C Passananti

10

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M Fanciulli

Cell Death & Differentiation

11

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language of work or name

English

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publication date

March 2008

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published in

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volume

15

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issue

3

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page(s)

515-20

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cites work

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TSG101 interacts with apoptosis-antagonizing transcription factor and enhances androgen receptor-mediated transcription by promoting its monoubiquitination

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Che-1 affects cell growth by interfering with the recruitment of HDAC1 by Rb

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AATF inhibits aberrant production of amyloid beta peptide 1-42 by interacting directly with Par-4

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Che-1 arrests human colon carcinoma cell proliferation by displacing HDAC1 from the p21WAF1/CIP1 promoter

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Che-1 phosphorylation by ATM/ATR and Chk2 kinases activates p53 transcription and the G2/M checkpoint

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Live or let die: the cell's response to p53

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IAP proteins: blocking the road to death's door

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Caspases: intracellular signaling by proteolysis

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Targeting XIAP for the treatment of malignancy

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The prolyl isomerase Pin1 affects Che-1 stability in response to apoptotic DNA damage

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NRAGE associates with the anti-apoptotic factor Che-1 and regulates its degradation to induce cell death

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Inhibition of JNK activation through NF-kappaB target genes

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IAP family proteins--suppressors of apoptosis

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The inhibitors of apoptosis (IAPs) and their emerging role in cancer

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Identification of a novel partner of RNA polymerase II subunit 11, Che-1, which interacts with and affects the growth suppression function of Rb

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7 January 2021