Mechanism of ASC-mediated apoptosis: bid-dependent apoptosis in type II cells (Q24302312)

positive regulation of extrinsic apoptotic signaling pathway

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

author name string

M Hasegawa

1

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K Kawase

2

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N Inohara

3

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R Imamura

4

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W-C Yeh

5

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T Kinoshita

6

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T Suda

Selective expression of FLIP in malignant melanocytic skin lesions

7

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language of work or name

English

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publication date

15 March 2007

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published in

Oncogene

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volume

26

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issue

12

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page(s)

1748-56

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cites work

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Human CARD12 is a novel CED4/Apaf-1 family member that induces apoptosis

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ASC-mediated NF-kappaB activation leading to interleukin-8 production requires caspase-8 and is inhibited by CLARP.

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Fas ligand induces cell-autonomous NF-kappaB activation and interleukin-8 production by a mechanism distinct from that of tumor necrosis factor-alpha.

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Nod1, an Apaf-1-like activator of caspase-9 and nuclear factor-kappaB

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CLARP, a death effector domain-containing protein interacts with caspase-8 and regulates apoptosis

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Salmonella-induced caspase-2 activation in macrophages: a novel mechanism in pathogen-mediated apoptosis

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Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

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Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

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Differential activation of the inflammasome by caspase-1 adaptors ASC and Ipaf

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Innate immunity against Francisella tularensis is dependent on the ASC/caspase-1 axis

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The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta

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ASC is an activating adaptor for NF-kappa B and caspase-8-dependent apoptosis

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c-FLIP mediates resistance of Hodgkin/Reed-Sternberg cells to death receptor-induced apoptosis

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TMS1/ASC: the cancer connection

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Nod2, a Nod1/Apaf-1 family member that is restricted to monocytes and activates NF-kappaB

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ASC is a Bax adaptor and regulates the p53-Bax mitochondrial apoptosis pathway

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Identification of Ipaf, a human caspase-1-activating protein related to Apaf-1

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Caspase-1 activator Ipaf is a p53-inducible gene involved in apoptosis

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Two CD95 (APO-1/Fas) signaling pathways

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Bcl-2 and Bcl-xL inhibit CD95-mediated apoptosis by preventing mitochondrial release of Smac/DIABLO and subsequent inactivation of X-linked inhibitor-of-apoptosis protein

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Regulation of lymphocyte proliferation and death by FLIP.

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The role of mitochondrial factors in apoptosis: a Russian roulette with more than one bullet.

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PYPAF7, a novel PYRIN-containing Apaf1-like protein that regulates activation of NF-kappa B and caspase-1-dependent cytokine processing

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Requirement for Casper (c-FLIP) in regulation of death receptor-induced apoptosis and embryonic development

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Caspase 8 small interfering RNA prevents acute liver failure in mice

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