RING-dependent tumor suppression and G2/M arrest induced by the TRC8 hereditary kidney cancer gene (Q24305480)

ubiquitin-like protein transferase activity

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

1 reference

A Brauweiler

1

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K L Lorick

2

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J P Lee

3

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Y C Tsai

4

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D Chan

5

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A M Weissman

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H A Drabkin

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R M Gemmill

The ubiquitin proteolytic system: from a vague idea, through basic mechanisms, and onto human diseases and drug targeting

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language of work or name

English

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publication date

5 April 2007

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published in

Oncogene

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volume

26

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issue

16

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page(s)

2263-71

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cites work

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Hereditary Renal-Cell Carcinoma Associated with a Chromosomal Translocation

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The tumor autocrine motility factor receptor, gp78, is a ubiquitin protein ligase implicated in degradation from the endoplasmic reticulum

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The TRC8 hereditary kidney cancer gene suppresses growth and functions with VHL in a common pathway

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Growth suppression induced by the TRC8 hereditary kidney cancer gene is dependent upon JAB1/CSN5.

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The hereditary renal cell carcinoma 3;8 translocation fuses FHIT to a patched-related gene, TRC8

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Protein sensors for membrane sterols

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SREBPs: activators of the complete program of cholesterol and fatty acid synthesis in the liver

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SREBP pathway responds to sterols and functions as an oxygen sensor in fission yeast

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RING fingers mediate ubiquitin-conjugating enzyme (E2)-dependent ubiquitination

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Autocrine platelet-derived growth factor-dependent gene expression in glioblastoma cells is mediated largely by activation of the transcription factor sterol regulatory element binding protein and is associated with altered genotype and patient surv

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HIF activation identifies early lesions in VHL kidneys: evidence for site-specific tumor suppressor function in the nephron

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