Smad3 and Smad4 cooperate with c-Jun/c-Fos to mediate TGF-beta-induced transcription (Q24311810)

SMAD family member 4

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Fos proto-oncogene, AP-1 transcription factor subunit

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Jun proto-oncogene, AP-1 transcription factor subunit

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SMAD family member 2

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positive regulation of transcription, DNA-templated

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TGF-beta signalling from cell membrane to nucleus through SMAD proteins

author name string

Y Zhang

series ordinal

1

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X H Feng

series ordinal

2

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R Derynck

series ordinal

3

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language of work or name

English

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publication date

27 August 1998

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published in

Nature

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volume

394

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page(s)

909-13

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issue

6696

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cites work

1 reference

7 January 2021

Partnership between DPC4 and SMAD proteins in TGF-beta signalling pathways.

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7 January 2021

Receptor-associated Mad homologues synergize as effectors of the TGF-beta response

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7 January 2021

The tumor suppressor Smad4/DPC 4 as a central mediator of Smad function

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7 January 2021

Transcriptional activation by TGF beta 1 mediated by the dyad symmetry element (DSE) and the TPA responsive element (TRE).

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7 January 2021

Regulation of clusterin gene expression by transforming growth factor beta

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7 January 2021

Tissue factor gene transcription in serum-stimulated fibroblasts is mediated by recruitment of c-Fos into specific AP-1 DNA-binding complexes

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7 January 2021

TGF beta signals through a heteromeric protein kinase receptor complex

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7 January 2021

TGF-beta receptor-mediated signalling through Smad2, Smad3 and Smad4

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7 January 2021

The human leukemia oncogene bcr-abl abrogates the anchorage requirement but not the growth factor requirement for proliferation

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7 January 2021

Phorbol ester-inducible genes contain a common cis element recognized by a TPA-modulated trans-acting factor

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7 January 2021

Heteromeric and homomeric interactions correlate with signaling activity and functional cooperativity of Smad3 and Smad4/DPC4

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7 January 2021

The c-Fos protein interacts with c-Jun/AP-1 to stimulate transcription of AP-1 responsive genes

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7 January 2021

The tumor suppressor Smad4/DPC4 and transcriptional adaptor CBP/p300 are coactivators for smad3 in TGF-beta-induced transcriptional activation

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7 January 2021

Mutations increasing autoinhibition inactivate tumour suppressors Smad2 and Smad4.

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7 January 2021

A human Mad protein acting as a BMP-regulated transcriptional activator

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7 January 2021

Transforming growth factor-beta (TGF-beta)-induced down-regulation of cyclin A expression requires a functional TGF-beta receptor complex. Characterization of chimeric and truncated type I and type II receptors

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7 January 2021

Down-regulation of the cyclin A promoter by transforming growth factor-beta1 is associated with a reduction in phosphorylated activating transcription factor-1 and cyclic AMP-responsive element-binding protein

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7 January 2021

Tumor suppressor Smad4 is a transforming growth factor beta-inducible DNA binding protein

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7 January 2021

Human Smad3 and Smad4 are sequence-specific transcription activators

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7 January 2021

The regulation of AP-1 activity by mitogen-activated protein kinases

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7 January 2021

Transformation suppressor activity of a Jun transcription factor lacking its activation domain

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7 January 2021

Opposing BMP and EGF signalling pathways converge on the TGF-beta family mediator Smad1

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7 January 2021

Smad2 transduces common signals from receptor serine-threonine and tyrosine kinases

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7 January 2021

Human transforming growth factor-beta 3: recombinant expression, purification, and biological activities in comparison with transforming growth factors-beta 1 and -beta 2.

1 reference

7 January 2021

Ligand-independent activation of transforming growth factor (TGF) beta signaling pathways by heteromeric cytoplasmic domains of TGF-beta receptors

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7 January 2021

Identifiers

DOI

10.1038/29814

1 reference

Consolidated OpenCitations Corpus – April 2017

Dimensions Publication ID

2682635

1019328835

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2682635

1 reference

Consolidated OpenCitations Corpus – April 2017

Consolidated OpenCitations Corpus – April 2017

1 reference