Complement C1q activates canonical Wnt signaling and promotes aging-related phenotypes (Q24337767)

GOA release 2020-03-11

23

Marina Botto

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Issei Komuro

25

Issei Komuro

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7

Katsuki Okada

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Ichiro Shiojima

24

Ichiro Shiojima

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Taku Sakai

8

Taku Sakai

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Tomoaki Higo

5

Tomoaki Higo

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Seitaro Nomura

3

Seitaro Nomura

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Tomokazu Sumida

2

Tomokazu Sumida

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Akemi Katada

14

Akemi Katada

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Akihito Hashimoto

9

Akihito Hashimoto

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Haruhiro Toko

13

Haruhiro Toko

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Hiroshi Akazawa

15

Hiroshi Akazawa

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author name string

Atsuhiko T Naito

1

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Mei-Lan Liu

4

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Akito Nakagawa

6

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Yurina Hara

10

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Ippei Shimizu

11

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Weidong Zhu

12

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Toru Oka

16

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Jong-Kook Lee

17

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Tohru Minamino

18

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Toshio Nagai

19

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Kenneth Walsh

20

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Akira Kikuchi

21

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Misako Matsumoto

Wnt/beta-catenin signaling: components, mechanisms, and diseases

22

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language of work or name

English

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publication date

8 June 2012

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published in

Cell

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volume

149

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issue

6

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page(s)

1298-313

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cites work

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Increased Wnt signaling during aging alters muscle stem cell fate and increases fibrosis

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Initiation of Wnt signaling: control of Wnt coreceptor Lrp6 phosphorylation/activation via frizzled, dishevelled and axin functions.

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p53-induced inhibition of Hif-1 causes cardiac dysfunction during pressure overload

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Targeted disruption of the homeobox transcription factor Bapx1 results in lethal skeletal dysplasia with asplenia and gastroduodenal malformation.

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A member of the Frizzled protein family mediating axis induction by Wnt-5A

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Ca2+/calmodulin-dependent kinase IIdelta causes heart failure by accumulation of p53 in dilated cardiomyopathy

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Identifiers

DOI

1 reference

823137

823137

1 reference

1 reference

1 reference