Versatile myeloid cell subsets contribute to tuberculosis-associated inflammation (Q26997007)

Stefan H.E. Kaufmann

series ordinal

2

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language of work or name

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August 2015

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European Journal of Immunology

published in

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volume

45

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page(s)

2191-202

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issue

8

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Robert Koch, the Nobel Prize, and the ongoing threat of tuberculosis

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The formation of the granuloma in tuberculosis infection

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Mycobacterium tuberculosis: success through dormancy

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A novel mycolic acid cyclopropane synthetase is required for cording, persistence, and virulence of Mycobacterium tuberculosis

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Regulation of Mycobacterium tuberculosis cell envelope composition and virulence by intramembrane proteolysis

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Diversity in Mycobacterium tuberculosis mannosylated cell wall determinants impacts adaptation to the host

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How sisters grow apart: mycobacterial growth and division

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Mycobacterium tuberculosis and the intimate discourse of a chronic infection

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Prevention of phagosome-lysosome fusion in cultured macrophages by sulfatides of Mycobacterium tuberculosis

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Cord factor trehalose 6,6'-dimycolate (TDM) mediates trafficking events during mycobacterial infection of murine macrophages

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The human macrophage mannose receptor directs Mycobacterium tuberculosis lipoarabinomannan-mediated phagosome biogenesis

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Manipulation of the mononuclear phagocyte system by Mycobacterium tuberculosis

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Nitric oxide synthase in innate and adaptive immunity: an update.

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Innate immune recognition of Mycobacterium tuberculosis

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Cell-autonomous effector mechanisms against mycobacterium tuberculosis

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Pro- and anti-inflammatory cytokines in tuberculosis: a two-edged sword in TB pathogenesis.

21 January 2018

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Cytokines in the balance of protection and pathology during mycobacterial infections

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Fatal Mycobacterium tuberculosis infection despite adaptive immune response in the absence of MyD88.

21 January 2018

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Caspase-1 independent IL-1beta production is critical for host resistance to mycobacterium tuberculosis and does not require TLR signaling in vivo

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Coactivation of Syk kinase and MyD88 adaptor protein pathways by bacteria promotes regulatory properties of neutrophils.

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The adaptor molecule CARD9 is essential for tuberculosis control

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Tumor necrosis factor alpha in mycobacterial infection

21 January 2018

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MyD88-deficient mice display a profound loss in resistance to Mycobacterium tuberculosis associated with partially impaired Th1 cytokine and nitric oxide synthase 2 expression

21 January 2018

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The role of IL-10 in immune regulation during M. tuberculosis infection

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Nucleotide-binding oligomerization domain protein 2-deficient mice control infection with Mycobacterium tuberculosis

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Nitric oxide controls the immunopathology of tuberculosis by inhibiting NLRP3 inflammasome-dependent processing of IL-1β.

21 January 2018

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NALP3 is not necessary for early protection against experimental tuberculosis.

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Granuloma formation and host defense in chronic Mycobacterium tuberculosis infection requires PYCARD/ASC but not NLRP3 or caspase-1.

21 January 2018

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Mycobacterium tuberculosis and the host cell inflammasome: a complex relationship

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Critical role of AIM2 in Mycobacterium tuberculosis infection

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Cutting edge: Mycobacterium tuberculosis but not nonvirulent mycobacteria inhibits IFN-β and AIM2 inflammasome-dependent IL-1β production via its ESX-1 secretion system

21 January 2018

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A bacterial cyclic dinucleotide activates the cytosolic surveillance pathway and mediates innate resistance to tuberculosis.

21 January 2018

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Mycobacterium tuberculosis activates the DNA-dependent cytosolic surveillance pathway within macrophages

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Mycobacterium tuberculosis Differentially Activates cGAS- and Inflammasome-Dependent Intracellular Immune Responses through ESX-1.

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The Cytosolic Sensor cGAS Detects Mycobacterium tuberculosis DNA to Induce Type I Interferons and Activate Autophagy

21 January 2018

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Cyclic GMP-AMP Synthase Is an Innate Immune DNA Sensor for Mycobacterium tuberculosis

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Extracellular M. tuberculosis DNA targets bacteria for autophagy by activating the host DNA-sensing pathway

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The DNA damage-regulated autophagy modulator DRAM1 links mycobacterial recognition via TLR-MYD88 to autophagic defense [corrected].

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Autophagy in tuberculosis

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AhR sensing of bacterial pigments regulates antibacterial defence.

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Role of innate cytokines in mycobacterial infection.

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IL-1β promotes antimicrobial immunity in macrophages by regulating TNFR signaling and caspase-3 activation

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Vitamin D induces interleukin-1β expression: paracrine macrophage epithelial signaling controls M. tuberculosis infection

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Host-directed therapy of tuberculosis based on interleukin-1 and type I interferon crosstalk

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iNKT cell production of GM-CSF controls Mycobacterium tuberculosis.

21 January 2018

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Host genetics of mycobacterial diseases in mice and men: forward genetic studies of BCG-osis and tuberculosis

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Killing of virulent Mycobacterium tuberculosis by reactive nitrogen intermediates produced by activated murine macrophages

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Reprogramming of the macrophage transcriptome in response to interferon-gamma and Mycobacterium tuberculosis: signaling roles of nitric oxide synthase-2 and phagocyte oxidase

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Vitamin D is required for IFN-gamma-mediated antimicrobial activity of human macrophages

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Immune Control of Tuberculosis by IFN-γ-Inducible LRG-47

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Autophagy is a defense mechanism inhibiting BCG and Mycobacterium tuberculosis survival in infected macrophages

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Interferon-gamma-treated murine macrophages inhibit growth of tubercle bacilli via the generation of reactive nitrogen intermediates

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IFN-inducible GTPases in host cell defense

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Interferon gamma activated macrophages kill mycobacteria by nitric oxide induced apoptosis

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T helper 2 cytokines inhibit autophagic control of intracellular Mycobacterium tuberculosis.

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Alternative activation deprives macrophages of a coordinated defense program to Mycobacterium tuberculosis.

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Cutting edge: vitamin D-mediated human antimicrobial activity against Mycobacterium tuberculosis is dependent on the induction of cathelicidin

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Toll-like receptor triggering of a vitamin D-mediated human antimicrobial response

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Type I interferon suppresses type II interferon-triggered human anti-mycobacterial responses

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Mycobacterium tuberculosis triggers host type I IFN signaling to regulate IL-1β production in human macrophages

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Innate and adaptive interferons suppress IL-1α and IL-1β production by distinct pulmonary myeloid subsets during Mycobacterium tuberculosis infection

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Type I IFN signaling triggers immunopathology in tuberculosis-susceptible mice by modulating lung phagocyte dynamics

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Dynamic roles of type I and type II IFNs in early infection with Mycobacterium tuberculosis

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Intranasal Poly-IC treatment exacerbates tuberculosis in mice through the pulmonary recruitment of a pathogen-permissive monocyte/macrophage population

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Resistance and susceptibility to tuberculosis analysed at the transcriptome level: lessons from mouse macrophages.

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Probing host pathogen cross-talk by transcriptional profiling of both Mycobacterium tuberculosis and infected human dendritic cells and macrophages

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Infection of human macrophages and dendritic cells with Mycobacterium tuberculosis induces a differential cytokine gene expression that modulates T cell response

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Mycobacterium tuberculosis infects dendritic cells with high frequency and impairs their function in vivo.

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Cell-to-cell transfer of M. tuberculosis antigens optimizes CD4 T cell priming

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Communication between Human Dendritic Cell Subsets in Tuberculosis: Requirements for Naive CD4(+) T Cell Stimulation

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Susceptibility to tuberculosis is associated with variants in the ASAP1 gene encoding a regulator of dendritic cell migration

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Foamy macrophages and the progression of the human tuberculosis granuloma

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Intracellular bacillary burden reflects a burst size for Mycobacterium tuberculosis in vivo.

21 January 2018

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Mycobacterium tuberculosis inhibits neutrophil apoptosis, leading to delayed activation of naive CD4 T cells

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Failure to recruit anti-inflammatory CD103+ dendritic cells and a diminished CD4+ Foxp3+ regulatory T cell pool in mice that display excessive lung inflammation and increased susceptibility to Mycobacterium tuberculosis

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Cytosolic access of Mycobacterium tuberculosis: critical impact of phagosomal acidification control and demonstration of occurrence in vivo

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Loss of RD1 contributed to the attenuation of the live tuberculosis vaccines Mycobacterium bovis BCG and Mycobacterium microti

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Neutrophils are the predominant infected phagocytic cells in the airways of patients with active pulmonary TB.

21 January 2018

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Reverse translation in tuberculosis: neutrophils provide clues for understanding development of active disease

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Neutrophils in tuberculosis: friend or foe?

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S100A8/A9 proteins mediate neutrophilic inflammation and lung pathology during tuberculosis

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CXCL5-secreting pulmonary epithelial cells drive destructive neutrophilic inflammation in tuberculosis

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Macrophages acquire neutrophil granules for antimicrobial activity against intracellular pathogens

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Neutrophil-mediated innate immune resistance to mycobacteria

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Lung neutrophils facilitate activation of naive antigen-specific CD4+ T cells during Mycobacterium tuberculosis infection

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Profiling early lung immune responses in the mouse model of tuberculosis

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Early granuloma formation after aerosol Mycobacterium tuberculosis infection is regulated by neutrophils via CXCR3-signaling chemokines.

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Genetically determined susceptibility to tuberculosis in mice causally involves accelerated and enhanced recruitment of granulocytes

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Neutrophil responses to Mycobacterium tuberculosis infection in genetically susceptible and resistant mice

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Damaging role of neutrophilic infiltration in a mouse model of progressive tuberculosis.

21 January 2018

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Platelets direct monocyte differentiation into epithelioid-like multinucleated giant foam cells with suppressive capacity upon mycobacterial stimulation

21 January 2018

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The neutrophil as a cellular source of chemokines

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MicroRNA-223 controls susceptibility to tuberculosis by regulating lung neutrophil recruitment

21 January 2018

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Interleukin 22 inhibits intracellular growth of Mycobacterium tuberculosis by enhancing calgranulin A expression.

21 January 2018

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DAMPs, PAMPs and alarmins: all we need to know about danger

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Regulation of neutrophils by interferon-γ limits lung inflammation during tuberculosis infection

21 January 2018

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Mycobacterium tuberculosis ESAT-6 is a leukocidin causing Ca2+ influx, necrosis and neutrophil extracellular trap formation

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Myeloid-derived suppressor cells as regulators of the immune system

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Increased frequency of myeloid-derived suppressor cells during active tuberculosis and after recent mycobacterium tuberculosis infection suppresses T-cell function

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Identification of CD244-expressing myeloid-derived suppressor cells in patients with active tuberculosis

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Gr-1dimCD11b+ immature myeloid-derived suppressor cells but not neutrophils are markers of lethal tuberculosis infection in mice

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Lung-residing myeloid-derived suppressors display dual functionality in murine pulmonary tuberculosis

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Gr1(int)CD11b+ myeloid-derived suppressor cells in Mycobacterium tuberculosis infection

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TNF dually mediates resistance and susceptibility to mycobacteria via mitochondrial reactive oxygen species

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Macrophage polarization drives granuloma outcome during Mycobacterium tuberculosis infection

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Computational modeling predicts IL-10 control of lesion sterilization by balancing early host immunity-mediated antimicrobial responses with caseation during mycobacterium tuberculosis infection

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Macrophage activation and polarization: nomenclature and experimental guidelines

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Macrophage arginase-1 controls bacterial growth and pathology in hypoxic tuberculosis granulomas

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Caseation of human tuberculosis granulomas correlates with elevated host lipid metabolism

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Inflammatory dendritic cells migrate in and out of transplanted chronic mycobacterial granulomas in mice

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Macrophage and T cell dynamics during the development and disintegration of mycobacterial granulomas

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Mycobacterium tuberculosis dysregulates MMP/TIMP balance to drive rapid cavitation and unrestrained bacterial proliferation

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Neutrophil-Derived MMP-8 Drives AMPK-Dependent Matrix Destruction in Human Pulmonary Tuberculosis

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Revisiting the role of the granuloma in tuberculosis

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Interferon-γ Regulates the Death of M. tuberculosis-Infected Macrophages.

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Paradoxical role of CD16+CCR2+CCR5+ monocytes in tuberculosis: efficient APC in pleural effusion but also mark disease severity in blood

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M. tuberculosis and M. leprae Translocate from the Phagolysosome to the Cytosol in Myeloid Cells

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Early lesion formation in cattle experimentally infected with Mycobacterium bovis

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Killing of Mycobacterium tuberculosis by neutrophils: a nonoxidative process

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https://api.crossref.org/works/10.1002%2FEJI.201545493

7 January 2021

Escape of Mycobacterium tuberculosis from oxidative killing by neutrophils

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201545493

7 January 2021

Identifiers

DOI

10.1002/EJI.201545493

1 reference

Consolidated OpenCitations Corpus – April 2017

4078792

4078792

1 reference

Consolidated OpenCitations Corpus – April 2017

Consolidated OpenCitations Corpus – April 2017

1 reference