TDP-43 identified from a genome wide RNAi screen for SOD1 regulators (Q28482611)

1

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Cameron E Day

2

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Shuguang Wei

3

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Philip J Thomas

Creative Commons Attribution 4.0 International

5

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language of work or name

English

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publication date

2012

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published in

PLOS One

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volume

7

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issue

4

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page(s)

e35818

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copyright license

start time

26 April 2012

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April 2022 Public Data File from Crossref

copyright status

copyrighted

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describes a project that uses

ImageQuant

object named as

Image Quant

1 reference

CZ Software Mentions: A large dataset of software mentions in the biomedical literature

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Amyotrophic lateral sclerosis

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Knockout of glutamate transporters reveals a major role for astroglial transport in excitotoxicity and clearance of glutamate

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A screen to identify cellular modulators of soluble levels of an amyotrophic lateral sclerosis (ALS)-causing mutant SOD1.

7 April 2017

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Long pre-mRNA depletion and RNA missplicing contribute to neuronal vulnerability from loss of TDP-43.

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TAR DNA-binding protein 43 (TDP-43) regulates stress granule dynamics via differential regulation of G3BP and TIA-1.

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TDP-43 is a transcriptional repressor: the testis-specific mouse acrv1 gene is a TDP-43 target in vivo

29 September 2017

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TDP-43 and FUS/TLS: emerging roles in RNA processing and neurodegeneration

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Cytoplasmic mislocalization of TDP-43 is toxic to neurons and enhanced by a mutation associated with familial amyotrophic lateral sclerosis

29 September 2017

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Global analysis of TDP-43 interacting proteins reveals strong association with RNA splicing and translation machinery.

29 September 2017

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Knockdown of transactive response DNA-binding protein (TDP-43) downregulates histone deacetylase 6

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TDP-43 is intrinsically aggregation-prone, and amyotrophic lateral sclerosis-linked mutations accelerate aggregation and increase toxicity

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Functional mapping of the interaction between TDP-43 and hnRNP A2 in vivo

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A novel CpG-free vertebrate insulator silences the testis-specific SP-10 gene in somatic tissues: role for TDP-43 in insulator function

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29 September 2017

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29 September 2017

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Evidence of increased oxidative damage in both sporadic and familial amyotrophic lateral sclerosis

2 June 2018

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Wild-Type Nonneuronal Cells Extend Survival of SOD1 Mutant Motor Neurons in ALS Mice

2 June 2018

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Pathological TDP-43 distinguishes sporadic amyotrophic lateral sclerosis from amyotrophic lateral sclerosis with SOD1 mutations.

21 January 2018

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50bp deletion in the promoter for superoxide dismutase 1 (SOD1) reduces SOD1 expression in vitro and may correlate with increased age of onset of sporadic amyotrophic lateral sclerosis

21 January 2018

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Mitochondrial dysfunction in neurodegenerative diseases

21 January 2018

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Mutations in dynein link motor neuron degeneration to defects in retrograde transport

21 January 2018

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Lack of TDP-43 abnormalities in mutant SOD1 transgenic mice shows disparity with ALS

21 January 2018

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Mitochondria and the pathogenesis of ALS

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Cell culture evidence for neuronal degeneration in amyotrophic lateral sclerosis being linked to glutamate AMPA/kainate receptors

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Increased 3-nitrotyrosine in both sporadic and familial amyotrophic lateral sclerosis

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Protein solubility and folding monitored in vivo by structural complementation of a genetic marker protein

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