Involvement of JAK2 upstream of the PI 3-kinase in cell-cell adhesion regulation by gastrin (Q28581791)

31 January 2017

title

Involvement of JAK2 upstream of the PI 3-kinase in cell-cell adhesion regulation by gastrin (English)

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31 January 2017

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Audrey Ferrand

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ORCID Public Data File 2021

Daniel Fourmy

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object named as

Daniel Fourmy

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31 January 2017

author name string

Aline Kowalski-Chauvel

2

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31 January 2017

Claudine Bertrand

3

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31 January 2017

Lucien Pradayrol

4

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31 January 2017

Marlene Dufresne

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31 January 2017

Catherine Seva

7

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31 January 2017

language of work or name

English

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publication date

10 December 2004

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Experimental Cell Research

31 January 2017

published in

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31 January 2017

volume

301

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31 January 2017

issue

2

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31 January 2017

page(s)

128–138

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The JAK/STAT signaling pathway

31 January 2017

cites work

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Gastrin mediated cholecystokinin-2 receptor activation induces loss of cell adhesion and scattering in epithelial MDCK cells

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Involvement of phosphatidylinositol 3-kinase and mitogen-activated protein kinases in glycine-extended gastrin-induced dissociation and migration of gastric epithelial cells.

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Stimulation of gastrin-CCKB receptor promotes migration of gastric AGS cells via multiple paracrine pathways

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Growth hormone and prolactin stimulate tyrosine phosphorylation of insulin receptor substrate-1, -2, and -3, their association with p85 phosphatidylinositol 3-kinase (PI3-kinase), and concomitantly PI3-kinase activation via JAK2 kinase

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Gastrin induces heparin-binding epidermal growth factor-like growth factor in rat gastric epithelial cells transfected with gastrin receptor

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Gastrin stimulates cyclooxygenase-2 expression in intestinal epithelial cells through multiple signaling pathways. Evidence for involvement of ERK5 kinase and transactivation of the epidermal growth factor receptor

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7 January 2021

Identifiers

DOI

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31 January 2017

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