PINK1 loss-of-function mutations affect mitochondrial complex I activity via NdufA10 ubiquinone uncoupling (Q28593164)

31 January 2017

title

PINK1 loss-of-function mutations affect mitochondrial complex I activity via NdufA10 ubiquinone uncoupling (English)

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NADH:ubiquinone oxidoreductase subunit A10

31 January 2017

main subject

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PTEN induced putative kinase 1

GOA release 2020-03-11

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PTEN-induced putative kinase 1 Dmel_CG4523

GOA release 2020-03-11

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GOA release 2020-03-11

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12

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Pieter-Jan De Bock

4

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Patrik Verstreken

13

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Liesbeth Aerts

object named as

Liesbeth Aerts

7

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Christine Klein

11

object named as

Christine Klein

1 reference

31 January 2017

14

Bart De Strooper

1 reference

31 January 2017

Vanessa A. Morais

1

1 reference

31 January 2017

Dominik Haddad

2

1 reference

31 January 2017

Katleen Craessaerts

3

1 reference

31 January 2017

Jef Swerts

5

1 reference

31 January 2017

Sven Vilain

6

1 reference

31 January 2017

Lut Overbergh

8

1 reference

31 January 2017

Anne Grünewald

9

1 reference

31 January 2017

Philip Seibler

10

1 reference

31 January 2017

language of work or name

English

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publication date

11 April 2014

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31 January 2017

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31 January 2017

volume

344

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31 January 2017

issue

6180

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31 January 2017

page(s)

203–207

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Hereditary early-onset Parkinson's disease caused by mutations in PINK1

31 January 2017

cites work

1 reference

https://api.crossref.org/works/10.1126/SCIENCE.1249161

PINK1 is selectively stabilized on impaired mitochondria to activate Parkin

21 April 2017

1 reference

https://api.crossref.org/works/10.1126/SCIENCE.1249161

21 April 2017

Mechanisms of mitophagy

1 reference

https://api.crossref.org/works/10.1126/SCIENCE.1249161

Bioenergetic consequences of PINK1 mutations in Parkinson disease

21 April 2017

1 reference

https://api.crossref.org/works/10.1126/SCIENCE.1249161

Loss of PINK1 causes mitochondrial functional defects and increased sensitivity to oxidative stress

21 April 2017

1 reference

https://api.crossref.org/works/10.1126/SCIENCE.1249161

p62/SQSTM1 is required for Parkin-induced mitochondrial clustering but not mitophagy; VDAC1 is dispensable for both

21 April 2017

1 reference

https://api.crossref.org/works/10.1126/SCIENCE.1249161

The yeast complex I equivalent NADH dehydrogenase rescues pink1 mutants

21 April 2017

1 reference

https://api.crossref.org/works/10.1126/SCIENCE.1249161

Mitochondrial Parkin recruitment is impaired in neurons derived from mutant PINK1 induced pluripotent stem cells

21 April 2017

1 reference

https://api.crossref.org/works/10.1126/SCIENCE.1249161

The architecture of respiratory complex I

21 April 2017

1 reference

https://api.crossref.org/works/10.1126/SCIENCE.1249161

Structural Basis for the Mechanism of Respiratory Complex I

21 April 2017

1 reference

https://api.crossref.org/works/10.1126/SCIENCE.1249161

OPA1 requires mitofusin 1 to promote mitochondrial fusion

21 April 2017

1 reference

https://api.crossref.org/works/10.1126/SCIENCE.1249161

Synaptojanin is recruited by endophilin to promote synaptic vesicle uncoating

21 April 2017

1 reference

https://api.crossref.org/works/10.1126/SCIENCE.1249161

Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin

21 April 2017

1 reference

https://api.crossref.org/works/10.1126%2FSCIENCE.1249161

Loss-of-function of human PINK1 results in mitochondrial pathology and can be rescued by parkin

21 April 2017

1 reference

https://api.crossref.org/works/10.1126%2FSCIENCE.1249161

Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin

21 April 2017

1 reference

https://api.crossref.org/works/10.1126%2FSCIENCE.1249161

Mitochondrial pathology and muscle and dopaminergic neuron degeneration caused by inactivation of Drosophila Pink1 is rescued by Parkin

21 April 2017

1 reference

https://api.crossref.org/works/10.1126%2FSCIENCE.1249161

The Parkinson's disease genes pink1 and parkin promote mitochondrial fission and/or inhibit fusion in Drosophila

21 April 2017

1 reference

https://api.crossref.org/works/10.1126%2FSCIENCE.1249161

Synaptic mitochondria are critical for mobilization of reserve pool vesicles at Drosophila neuromuscular junctions

21 April 2017

1 reference

https://api.crossref.org/works/10.1126%2FSCIENCE.1249161

PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy

21 April 2017

1 reference

https://api.crossref.org/works/10.1126%2FSCIENCE.1249161

A tissue-specific atlas of mouse protein phosphorylation and expression

21 April 2017

1 reference

https://api.crossref.org/works/10.1126%2FSCIENCE.1249161