Fragile X mental retardation protein targets G quartet mRNAs important for neuronal function (Q29616372)

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30 April 2017

author name string

Darnell JC

1

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30 April 2017

Jensen KB

2

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Jin P

3

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Brown V

4

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Warren ST

5

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Darnell RB

6

1 reference

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=11719189

30 April 2017

language of work or name

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30 April 2017

publication date

16 November 2001

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30 April 2017

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volume

107

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page(s)

489-99

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issue

4

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Dynamic visualization of local protein synthesis in hippocampal neurons

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The fragile X mental retardation protein is a ribonucleoprotein containing both nuclear localization and nuclear export signals

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FMRP associates with polyribosomes as an mRNP, and the I304N mutation of severe fragile X syndrome abolishes this association

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Human werner syndrome DNA helicase unwinds tetrahelical structures of the fragile X syndrome repeat sequence d(CGG)n

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Concerted activities of the RNA recognition and the glycine-rich C-terminal domains of nucleolin are required for efficient complex formation with pre-ribosomal RNA

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Ectopic expression of Hel-N1, an RNA-binding protein, increases glucose transporter (GLUT1) expression in 3T3-L1 adipocytes

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Identifiers

DOI

1 reference

Consolidated OpenCitations Corpus – April 2017

386246

386246

1 reference

Consolidated OpenCitations Corpus – April 2017

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=11719189

2 references

30 April 2017

Consolidated OpenCitations Corpus – April 2017