Highly conserved and disease-specific patterns of carboxyterminally truncated Abeta peptides 1-37/38/39 in addition to 1-40/42 in Alzheimer's disease and in patients with chronic neuroinflammation. (Q31078434)

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English	Highly conserved and disease-specific patterns of carboxyterminally truncated Abeta peptides 1-37/38/39 in addition to 1-40/42 in Alzheimer's disease and in patients with chronic neuroinflammation.	scientific article

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10 November 2019

Highly conserved and disease-specific patterns of carboxyterminally truncated Abeta peptides 1-37/38/39 in addition to 1-40/42 in Alzheimer's disease and in patients with chronic neuroinflammation (English)

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10 November 2019

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Alzheimer's disease

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10 November 2019

Johannes Kornhuber

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10 November 2019

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H Esselmann

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10 November 2019

M Bibl

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10 November 2019

A Smirnov

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10 November 2019

M Otto

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10 November 2019

S Paul

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10 November 2019

B Schmidt

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10 November 2019

H-W Klafki

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10 November 2019

M Maler

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10 November 2019

T Dyrks

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10 November 2019

M Bienert

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10 November 2019

M Beyermann

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10 November 2019

E Rüther

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1 May 2002

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10 November 2019

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Journal of Neurochemistry

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10 November 2019

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10 November 2019

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10 November 2019

481-496

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10 November 2019

Cerebrospinal fluid beta-amyloid(1-42) in Alzheimer disease: differences between early- and late-onset Alzheimer disease and stability during the course of disease.

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High cerebrospinal fluid tau and low amyloid beta42 levels in the clinical diagnosis of Alzheimer disease and relation to apolipoprotein E genotype.

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Amyloid beta-peptide is produced by cultured cells during normal metabolism

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The carboxy terminus of the beta amyloid protein is critical for the seeding of amyloid formation: implications for the pathogenesis of Alzheimer's disease

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Soluble amyloid beta peptide concentration as a predictor of synaptic change in Alzheimer's disease

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