Malaria parasite infection compromises control of concurrent systemic non-typhoidal Salmonella infection via IL-10-mediated alteration of myeloid cell function (Q33553714)

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English	Malaria parasite infection compromises control of concurrent systemic non-typhoidal Salmonella infection via IL-10-mediated alteration of myeloid cell function	scientific article

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scholarly article

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Europe PubMed Central

28 July 2017

Malaria parasite infection compromises control of concurrent systemic non-typhoidal Salmonella infection via IL-10-mediated alteration of myeloid cell function (English)

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Europe PubMed Central

28 July 2017

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Jason P. Mooney

object named as

Jason P Mooney

2

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Mariana Xavier Byndloss

object named as

Mariana N Xavier

4

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Renée M Tsolis

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Renée M Tsolis

10

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object named as

Werner Müller

8

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author name string

Kristen L Lokken

1

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Europe PubMed Central

28 July 2017

Brian P Butler

3

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Europe PubMed Central

28 July 2017

Jennifer Y Chau

5

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Europe PubMed Central

28 July 2017

Nicola Schaltenberg

6

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Europe PubMed Central

28 July 2017

Ramie H Begum

7

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Europe PubMed Central

28 July 2017

Shirley Luckhart

9

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Europe PubMed Central

28 July 2017

publication date

1 May 2014

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Europe PubMed Central

28 July 2017

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Europe PubMed Central

28 July 2017

10

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Europe PubMed Central

28 July 2017

5

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Europe PubMed Central

28 July 2017

e1004049

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Europe PubMed Central

28 July 2017

copyright license

Creative Commons Attribution 4.0 International

1 May 2014

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April 2022 Public Data File from Crossref

copyright status

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Salmonella require the fatty acid regulator PPARδ for the establishment of a metabolic environment essential for long-term persistence

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CD4+ T cell-derived IL-10 promotes Brucella abortus persistence via modulation of macrophage function

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Both hemolytic anemia and malaria parasite-specific factors increase susceptibility to Nontyphoidal Salmonella enterica serovar typhimurium infection in mice

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1 reference

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Salmonella infections in immunocompromised adults

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IL-10 from CD4CD25Foxp3CD127 adaptive regulatory T cells modulates parasite clearance and pathology during malaria infection

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Plasmodium chabaudi chabaudi: differential susceptibility of gene-targeted mice deficient in IL-10 to an erythrocytic-stage infection.

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T cell-specific inactivation of the interleukin 10 gene in mice results in enhanced T cell responses but normal innate responses to lipopolysaccharide or skin irritation

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Monocytes/macrophages and/or neutrophils are the target of IL-10 in the LPS endotoxemia model

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21 January 2018

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10.1371/JOURNAL.PPAT.1004049

1 reference

Europe PubMed Central

28 July 2017

1 reference

Europe PubMed Central

28 July 2017

1 reference

Europe PubMed Central

28 July 2017

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