In vivo disruption of TGF-beta signaling by Smad7 in airway epithelium alleviates allergic asthma but aggravates lung carcinogenesis in mouse (Q33559861)

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English	In vivo disruption of TGF-beta signaling by Smad7 in airway epithelium alleviates allergic asthma but aggravates lung carcinogenesis in mouse	scientific article

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scholarly article

1 reference

Europe PubMed Central

28 July 2017

In vivo disruption of TGF-beta signaling by Smad7 in airway epithelium alleviates allergic asthma but aggravates lung carcinogenesis in mouse (English)

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Europe PubMed Central

28 July 2017

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allergic asthma

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based on heuristic

inferred from title

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based on heuristic

inferred from title

object named as

Xiaolin Luo

1

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author name string

Qiurong Ding

2

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Europe PubMed Central

28 July 2017

Min Wang

3

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Europe PubMed Central

28 July 2017

Zhigang Li

4

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Europe PubMed Central

28 July 2017

Kairui Mao

5

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Europe PubMed Central

28 July 2017

Bing Sun

6

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Europe PubMed Central

28 July 2017

Yi Pan

7

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Europe PubMed Central

28 July 2017

Zhenzhen Wang

8

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Europe PubMed Central

28 July 2017

Ying Qin Zang

9

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Europe PubMed Central

28 July 2017

Yan Chen

10

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Europe PubMed Central

28 July 2017

language of work or name

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publication date

13 April 2010

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Europe PubMed Central

28 July 2017

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Europe PubMed Central

28 July 2017

5

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Europe PubMed Central

28 July 2017

4

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Europe PubMed Central

28 July 2017

e10149

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Europe PubMed Central

28 July 2017

copyright license

Creative Commons Attribution 4.0 International

13 April 2010

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April 2022 Public Data File from Crossref

copyright status

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Clara cell: progenitor for the bronchiolar epithelium

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SMAD6 contributes to patient survival in non-small cell lung cancer and its knockdown reestablishes TGF-beta homeostasis in lung cancer cells

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Epithelium: at the interface of innate and adaptive immune responses

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In vivo disruption of TGF-beta signaling by Smad7 leads to premalignant ductal lesions in the pancreas

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Prolonged allergen challenge in mice leads to persistent airway remodelling.

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Expression of Smad7 in bronchial epithelial cells is inversely correlated to basement membrane thickness and airway hyperresponsiveness in patients with asthma

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CD4(+) T helper cells engineered to produce latent TGF-beta1 reverse allergen-induced airway hyperreactivity and inflammation

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Airway remodeling in asthma

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Th2 cells and GATA-3 in asthma: new insights into the regulation of airway inflammation

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The MAD-related protein Smad7 associates with the TGFbeta receptor and functions as an antagonist of TGFbeta signaling

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Identifiers

10.1371/JOURNAL.PONE.0010149

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Europe PubMed Central

28 July 2017

2010PLoSO...510149L

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Europe PubMed Central

28 July 2017

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Europe PubMed Central

28 July 2017

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