MicroRNA 26a (miR-26a)/KLF4 and CREB-C/EBPβ regulate innate immune signaling, the polarization of macrophages and the trafficking of Mycobacterium tuberculosis to lysosomes during infection. (Q33783769)

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English	MicroRNA 26a (miR-26a)/KLF4 and CREB-C/EBPβ regulate innate immune signaling, the polarization of macrophages and the trafficking of Mycobacterium tuberculosis to lysosomes during infection.	scientific article

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21 March 2020

MicroRNA 26a (miR-26a)/KLF4 and CREB-C/EBPβ regulate innate immune signaling, the polarization of macrophages and the trafficking of Mycobacterium tuberculosis to lysosomes during infection (English)

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21 March 2020

Mycobacterium tuberculosis

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11

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21 March 2020

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Sanjaya Kumar Sahu

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21 March 2020

Manish Kumar

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21 March 2020

Sohini Chakraborty

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21 March 2020

Srijon Kaushik Banerjee

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21 March 2020

Ranjeet Kumar

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21 March 2020

Pushpa Gupta

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21 March 2020

Kuladip Jana

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21 March 2020

Umesh D Gupta

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21 March 2020

Zhumur Ghosh

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21 March 2020

Manikuntala Kundu

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30 May 2017

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21 March 2020

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21 March 2020

13

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21 March 2020

5

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21 March 2020

e1006410

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21 March 2020

copyright license

Creative Commons Attribution 4.0 International

30 May 2017

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April 2022 Public Data File from Crossref

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0 references

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Post-Transcriptional Regulation of KLF4 by High-Risk Human Papillomaviruses Is Necessary for the Differentiation-Dependent Viral Life Cycle.

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Differentiation-Dependent KLF4 Expression Promotes Lytic Epstein-Barr Virus Infection in Epithelial Cells

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Mycobacterium tuberculosis-Induced Polarization of Human Macrophage Orchestrates the Formation and Development of Tuberculous Granulomas In Vitro

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Transcription factors STAT6 and KLF4 implement macrophage polarization via the dual catalytic powers of MCPIP

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MicroRNA let-7 modulates the immune response to Mycobacterium tuberculosis infection via control of A20, an inhibitor of the NF-κB pathway.

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Krüppel-like factor 4 negatively regulates cellular antiviral immune response

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Mycobacterium tuberculosis decreases human macrophage IFN-γ responsiveness through miR-132 and miR-26a

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Macrophage arginase-1 controls bacterial growth and pathology in hypoxic tuberculosis granulomas

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Role of TNF in the altered interaction of dormant Mycobacterium tuberculosis with host macrophages.

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Krüppel-like factor 6 is a co-activator of NF-κB that mediates p65-dependent transcription of selected downstream genes

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Kruppel-like transcription factor 6 regulates inflammatory macrophage polarization

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4 July 2018

miR-16 and miR-26a target checkpoint kinases Wee1 and Chk1 in response to p53 activation by genotoxic stress

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MicroRNA-223 controls susceptibility to tuberculosis by regulating lung neutrophil recruitment

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Nitric oxide and KLF4 protein epigenetically modify class II transactivator to repress major histocompatibility complex II expression during Mycobacterium bovis bacillus Calmette-Guerin infection

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Decreased expression of miR-21, miR-26a, miR-29a, and miR-142-3p in CD4⁺ T cells and peripheral blood from tuberculosis patients

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CCAAT/enhancer-binding protein β (C/EBPβ) expression regulates dietary-induced inflammation in macrophages and adipose tissue in mice

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Transcriptional regulation of macrophage polarization: enabling diversity with identity

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Mycobacterium tuberculosis lipomannan blocks TNF biosynthesis by regulating macrophage MAPK-activated protein kinase 2 (MK2) and microRNA miR-125b.

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Krüppel-like factor 4 regulates macrophage polarization

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A CREB-C/EBPbeta cascade induces M2 macrophage-specific gene expression and promotes muscle injury repair

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Toll-like receptor-induced arginase 1 in macrophages thwarts effective immunity against intracellular pathogens

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RNA-binding proteins and post-transcriptional gene regulation

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4 July 2018

MicroRNA-26a targets the histone methyltransferase Enhancer of Zeste homolog 2 during myogenesis

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Autophagy in immune defense against Mycobacterium tuberculosis

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4 July 2018

Autophagy is a defense mechanism inhibiting BCG and Mycobacterium tuberculosis survival in infected macrophages

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4 July 2018

MicroRNAs: small RNAs with a big role in gene regulation

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4 July 2018

Enhancer-mediated control of macrophage-specific arginase I expression

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4 July 2018

Specificity of microRNA target selection in translational repression

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4 July 2018

Role of CREB in transcriptional regulation of CCAAT/enhancer-binding protein beta gene during adipogenesis

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4 July 2018

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4 July 2018

C/EBPbeta regulation in lipopolysaccharide-stimulated macrophages

1 reference

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4 July 2018

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1 reference

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4 July 2018

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4 July 2018

Alternative activation deprives macrophages of a coordinated defense program to Mycobacterium tuberculosis.

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Identification of a novel role of ESAT-6-dependent miR-155 induction during infection of macrophages with Mycobacterium tuberculosis.

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21 January 2018

Determination of arginase activity in macrophages: a micromethod

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Tuberculosis is associated with a down-modulatory lung immune response that impairs Th1-type immunity

1 reference

https://pubmed.ncbi.nlm.nih.gov/28558034

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

A liver-enriched transcriptional activator protein, LAP, and a transcriptional inhibitory protein, LIP, are translated from the same mRNA

1 reference

https://pubmed.ncbi.nlm.nih.gov/28558034

12 December 2020

based on heuristic

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10.1371/JOURNAL.PPAT.1006410

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21 March 2020

release_zhotxds7t5bfvolv7wpqqtokca

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24 November 2022

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21 March 2020

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21 March 2020

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