Blocking the class I histone deacetylase ameliorates renal fibrosis and inhibits renal fibroblast activation via modulating TGF-beta and EGFR signaling (Q34559028)

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English	Blocking the class I histone deacetylase ameliorates renal fibrosis and inhibits renal fibroblast activation via modulating TGF-beta and EGFR signaling	scientific article

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scholarly article

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Europe PubMed Central

3 August 2017

Blocking the class I histone deacetylase ameliorates renal fibrosis and inhibits renal fibroblast activation via modulating TGF-beta and EGFR signaling (English)

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Europe PubMed Central

3 August 2017

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Murugavel Ponnusamy

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Murugavel Ponnusamy

4

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author name string

Na Liu

1

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Europe PubMed Central

3 August 2017

Song He

2

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Europe PubMed Central

3 August 2017

Li Ma

3

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Europe PubMed Central

3 August 2017

Jinhua Tang

5

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Europe PubMed Central

3 August 2017

Evelyn Tolbert

6

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Europe PubMed Central

3 August 2017

George Bayliss

7

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Europe PubMed Central

3 August 2017

Ting C Zhao

8

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Europe PubMed Central

3 August 2017

Haidong Yan

9

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Europe PubMed Central

3 August 2017

Shougang Zhuang

10

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Europe PubMed Central

3 August 2017

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16 January 2013

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Europe PubMed Central

3 August 2017

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Europe PubMed Central

3 August 2017

8

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Europe PubMed Central

3 August 2017

1

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Europe PubMed Central

3 August 2017

e54001

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Europe PubMed Central

3 August 2017

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Creative Commons Attribution 4.0 International

16 January 2013

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April 2022 Public Data File from Crossref

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Genetic or pharmacologic blockade of EGFR inhibits renal fibrosis.

1 reference

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7 July 2018

Selective class I histone deacetylase inhibition suppresses hypoxia-induced cardiopulmonary remodeling through an antiproliferative mechanism

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7 July 2018

Role of TGF-β in chronic kidney disease: an integration of tubular, glomerular and vascular effects

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7 July 2018

EGFR signaling promotes TGFβ-dependent renal fibrosis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3546966

7 July 2018

Cellular and molecular mechanisms of renal fibrosis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3546966

7 July 2018

Diverse roles of TGF-β/Smads in renal fibrosis and inflammation

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Valproic acid attenuates proteinuria and kidney injury

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Transforming growth factor-β and Smads

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Suramin inhibits renal fibrosis in chronic kidney disease.

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Histone deacetylase inhibition attenuates diabetes-associated kidney growth: potential role for epigenetic modification of the epidermal growth factor receptor

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Acetylation modulates prolactin receptor dimerization

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Renal fibrosis: novel insights into mechanisms and therapeutic targets.

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Histone deacetylase: a potential therapeutic target for fibrotic disorders

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Nonhistone protein acetylation as cancer therapy targets

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A novel STAT3 inhibitor, S3I-201, attenuates renal interstitial fibroblast activation and interstitial fibrosis in obstructive nephropathy

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Epithelial cell cycle arrest in G2/M mediates kidney fibrosis after injury

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The microtubule-associated histone deacetylase 6 (HDAC6) regulates epidermal growth factor receptor (EGFR) endocytic trafficking and degradation

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Novel histone deacetylase inhibitors in clinical trials as anti-cancer agents

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Regulation of epidermal growth factor receptor trafficking by lysine deacetylase HDAC6

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Inhibition of histone deacetylase activity attenuates renal fibroblast activation and interstitial fibrosis in obstructive nephropathy.

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Histone deacetylase inhibition and the regulation of cell growth with particular reference to liver pathobiology

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Clinical studies of histone deacetylase inhibitors.

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Histone deacetylase inhibitor KBH-A42 inhibits cytokine production in RAW 264.7 macrophage cells and in vivo endotoxemia model

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Histone deacetylases as transducers and targets of nuclear signaling

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Cellular and molecular mechanisms of fibrosis

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Acetylation-dependent signal transduction for type I interferon receptor

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TGF-beta in renal injury and disease

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Hdac2 regulates the cardiac hypertrophic response by modulating Gsk3 beta activity

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Renal fibrosis: new insights into the pathogenesis and therapeutics

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Control of cardiac growth by histone acetylation/deacetylation

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Stat3 dimerization regulated by reversible acetylation of a single lysine residue

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Suppression of histone deacetylases worsens right ventricular dysfunction after pulmonary artery banding in rats

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27 October 2018

Identifiers

10.1371/JOURNAL.PONE.0054001

1 reference

Europe PubMed Central

3 August 2017

2013PLoSO...854001L

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1 reference

Europe PubMed Central

3 August 2017

1 reference

Europe PubMed Central

3 August 2017

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