NF-kappaB-mediated modulation of inducible nitric oxide synthase activity controls induction of the Epstein-Barr virus productive cycle by transforming growth factor beta 1 (Q35077613)

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English	NF-kappaB-mediated modulation of inducible nitric oxide synthase activity controls induction of the Epstein-Barr virus productive cycle by transforming growth factor beta 1	scientific article

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scholarly article

1 reference

Europe PubMed Central

6 August 2017

NF-kappaB-mediated modulation of inducible nitric oxide synthase activity controls induction of the Epstein-Barr virus productive cycle by transforming growth factor beta 1 (English)

1 reference

Europe PubMed Central

6 August 2017

Epstein–Barr virus

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author name string

Lassad Oussaief

1

1 reference

Europe PubMed Central

6 August 2017

Vanessa Ramírez

2

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Europe PubMed Central

6 August 2017

Aurélie Hippocrate

3

1 reference

Europe PubMed Central

6 August 2017

Hratch Arbach

4

1 reference

Europe PubMed Central

6 August 2017

Chantal Cochet

5

1 reference

Europe PubMed Central

6 August 2017

Alexis Proust

6

1 reference

Europe PubMed Central

6 August 2017

Martine Raphaël

7

1 reference

Europe PubMed Central

6 August 2017

Ridha Khelifa

8

1 reference

Europe PubMed Central

6 August 2017

Irène Joab

9

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Europe PubMed Central

6 August 2017

language of work or name

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publication date

20 April 2011

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Europe PubMed Central

6 August 2017

Journal of Virology

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Europe PubMed Central

6 August 2017

85

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Europe PubMed Central

6 August 2017

6502-6512

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Europe PubMed Central

6 August 2017

13

1 reference

Europe PubMed Central

6 August 2017

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1 reference

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Binding of NF-kappaB p65 subunit to the promoter elements is involved in LPS-induced transactivation of miRNA genes in human biliary epithelial cells

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Inducible nitric-oxide synthase expression is regulated by mitogen-activated protein kinase phosphatase-1.

1 reference

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Phosphatidylinositol 3-kinase/Akt pathway targets acetylation of Smad3 through Smad3/CREB-binding protein interaction: contribution to transforming growth factor beta1-induced Epstein-Barr virus reactivation

1 reference

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Activation of the lytic program of the Epstein-Barr virus in Burkitt's lymphoma cells leads to a two steps downregulation of expression of the proapoptotic protein BimEL, one of which is EBV-late-gene expression dependent

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Ribosomal protein S3: a KH domain subunit in NF-kappaB complexes that mediates selective gene regulation

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LMP1 TRAFficking activates growth and survival pathways

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NRAS mutation causes a human autoimmune lymphoproliferative syndrome.

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Virus and cell RNAs expressed during Epstein-Barr virus replication

1 reference

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Epstein-Barr virus (EBV) genome and expression in breast cancer tissue: effect of EBV infection of breast cancer cells on resistance to paclitaxel (Taxol)

1 reference

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Genome-wide transcription program and expression of the Rta responsive gene of Epstein-Barr virus

1 reference

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Role of Smad proteins in the regulation of NF-kappaB by TGF-beta in colon cancer cells.

1 reference

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Effects of mechanistically distinct NF-kappaB inhibitors on glial inducible nitric-oxide synthase expression

1 reference

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Identification of microRNAs of the herpesvirus family

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Dual role for TGF-beta1 in apoptosis

1 reference

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Down-regulation of inducible nitric oxide synthase by lysophosphatidic acid in human respiratory epithelial cells

1 reference

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The Epstein-Barr virus transactivator Zta binds to its own promoter and is required for full promoter activity during anti-Ig and TGF-beta1 mediated reactivation

1 reference

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Activation of Epstein-Barr virus by saliva from Sjogren's syndrome patients

1 reference

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Epstein-Barr virus and cancer

1 reference

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Activin receptor-like kinase (ALK)1 is an antagonistic mediator of lateral TGFbeta/ALK5 signaling

1 reference

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Two carboxyl-terminal activation regions of Epstein-Barr virus latent membrane protein 1 activate NF-kappaB through distinct signaling pathways in fibroblast cell lines

1 reference

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Mechanisms of TGF-beta signaling from cell membrane to the nucleus

1 reference

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Activation of NF-kappa B in virus-infected macrophages is dependent on mitochondrial oxidative stress and intracellular calcium: downstream involvement of the kinases TGF-beta-activated kinase 1, mitogen-activated kinase/extracellular signal-regulat

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Induction of macrophage nitric oxide production by Gram-negative flagellin involves signaling via heteromeric Toll-like receptor 5/Toll-like receptor 4 complexes

1 reference

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NF-kappaB and virus infection: who controls whom

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12-O-tetradecanoylphorbol-13-acetate induces Epstein-Barr virus reactivation via NF-kappaB and AP-1 as regulated by protein kinase C and mitogen-activated protein kinase

1 reference

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Activators of the Epstein-Barr virus lytic program concomitantly induce apoptosis, but lytic gene expression protects from cell death

1 reference

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Hostile takeovers: viral appropriation of the NF-kB pathway

1 reference

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Transforming growth factor beta 1 stimulates expression of the Epstein-Barr virus BZLF1 immediate-early gene product ZEBRA by an indirect mechanism which requires the MAPK kinase pathway

1 reference

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Transcriptional control by the TGF-beta/Smad signaling system

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1 reference

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1 reference

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The bZIP transactivator of Epstein-Barr virus, BZLF1, functionally and physically interacts with the p65 subunit of NF-kappa B

1 reference

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Mechanisms of suppression of macrophage nitric oxide release by transforming growth factor beta

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3126544

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Nitric oxide produced by human B lymphocytes inhibits apoptosis and Epstein-Barr virus reactivation

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3126544

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The Epstein-Barr virus early protein EB1 activates transcription from different responsive elements including AP-1 binding sites

1 reference

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Endogenous TGF-beta contributes to the induction of the EBV lytic cycle in two Burkitt lymphoma cell lines

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12 December 2020

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https://pubmed.ncbi.nlm.nih.gov/21507981

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1 reference

https://pubmed.ncbi.nlm.nih.gov/21507981

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based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1128/JVI.02560-10

1 reference

Europe PubMed Central

6 August 2017

1 reference

Europe PubMed Central

6 August 2017

1 reference

Europe PubMed Central

6 August 2017

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