Aberrant epigenetic and genetic marks are seen in myelodysplastic leukocytes and reveal Dock4 as a candidate pathogenic gene on chromosome 7q (Q35107545)

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English	Aberrant epigenetic and genetic marks are seen in myelodysplastic leukocytes and reveal Dock4 as a candidate pathogenic gene on chromosome 7q	scientific article

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scholarly article

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Europe PubMed Central

6 August 2017

Aberrant epigenetic and genetic marks are seen in myelodysplastic leukocytes and reveal Dock4 as a candidate pathogenic gene on chromosome 7q (English)

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Europe PubMed Central

6 August 2017

Maria E Figueroa

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Maria Figueroa

9

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Andrea Pellagatti

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Andrea Pellagatti

18

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Melissa Fazzari

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Melissa Fazzari

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Jacqueline Boultwood

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Jacqueline Boultwood

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Omar Abdel-Wahab

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Omar Abdel-Wahab

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Tushar D Bhagat

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Tushar Bhagat

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20

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Ulrich Steidl

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3

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Davendra Sohal

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27

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Ari Melnick

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6 August 2017

Cristina Alencar

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Cristina Alencar

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6 August 2017

Suman Kambhampati

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Suman Kambhampati

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13

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Simrit Parmar

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Christine M McMahon

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Christine McMahon

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6 August 2017

Leonidas C Platanias

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Leonidas C Platanias

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6 August 2017

Amittha Wickrema

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Amittha Wickrema

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29

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John M Greally

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6 August 2017

author name string

Li Zhou

1

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6 August 2017

Joanna Opalinska

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6 August 2017

Yiting Yu

4

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6 August 2017

Yongkai Mo

5

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6 August 2017

Jinghang Zhang

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6 August 2017

Sangeeta Nischal

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6 August 2017

Christoph Hueck

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6 August 2017

Masako Suzuki

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6 August 2017

Ellen Freidman

17

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6 August 2017

Yogen Sauthararajah

21

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6 August 2017

Vijay Yajnik

22

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6 August 2017

Steven D Gore

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Ross Levine

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6 August 2017

Amit Verma

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6 August 2017

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publication date

30 April 2011

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6 August 2017

Journal of Biological Chemistry

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6 August 2017

286

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6 August 2017

28

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6 August 2017

25211-25223

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6 August 2017

Widespread hypomethylation occurs early and synergizes with gene amplification during esophageal carcinogenesis

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Leukemic IDH1 and IDH2 mutations result in a hypermethylation phenotype, disrupt TET2 function, and impair hematopoietic differentiation

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DNA methylation signatures identify biologically distinct subtypes in acute myeloid leukemia.

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MDS and secondary AML display unique patterns and abundance of aberrant DNA methylation

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Genome-wide epigenetic analysis delineates a biologically distinct immature acute leukemia with myeloid/T-lymphoid features.

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Aberrant DNA methylation is a dominant mechanism in MDS progression to AML.

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A network of Krüppel-like Factors (Klfs). Klf8 is repressed by Klf3 and activated by Klf1 in vivo

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Molecular association between beta-catenin degradation complex and Rac guanine exchange factor DOCK4 is essential for Wnt/beta-catenin signaling

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Inhibition of the TGF-beta receptor I kinase promotes hematopoiesis in MDS.

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An integrative genomic and epigenomic approach for the study of transcriptional regulation

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An analytical pipeline for genomic representations used for cytosine methylation studies

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Chromosomal lesions and uniparental disomy detected by SNP arrays in MDS, MDS/MPD, and MDS-derived AML

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Results of a randomized study of 3 schedules of low-dose decitabine in higher-risk myelodysplastic syndrome and chronic myelomonocytic leukemia

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9 July 2018

Comparative isoschizomer profiling of cytosine methylation: the HELP assay

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9 July 2018

Microarray-based DNA methylation profiling: technology and applications

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9 July 2018

Finer delineation and transcript map of the 7q31 locus deleted in myeloid neoplasms

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Gene set enrichment analysis: a knowledge-based approach for interpreting genome-wide expression profiles

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Functional analysis of human hematopoietic stem cell gene expression using zebrafish

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Chromosome-wide and promoter-specific analyses identify sites of differential DNA methylation in normal and transformed human cells

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DOCK4, a GTPase activator, is disrupted during tumorigenesis

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Worms gang up on bacteria

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Gene Expression Omnibus: NCBI gene expression and hybridization array data repository

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9 July 2018

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https://api.crossref.org/works/10.1074%2FJBC.M111.235028

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Phase 1 study of low-dose prolonged exposure schedules of the hypomethylating agent 5-aza-2'-deoxycytidine (decitabine) in hematopoietic malignancies

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High-resolution whole genome tiling path array CGH analysis of CD34+ cells from patients with low-risk myelodysplastic syndromes reveals cryptic copy number alterations and predicts overall and leukemia-free survival.

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Gene-expression profiling of CD34+ hematopoietic cells expanded in a collagen I matrix.

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Evidence for reduced B-cell progenitors in early (low-risk) myelodysplastic syndrome.

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Activation of the p38 mitogen-activated protein kinase mediates the suppressive effects of type I interferons and transforming growth factor-beta on normal hematopoiesis

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Cutting edge: activation of the p38 mitogen-activated protein kinase signaling pathway mediates cytokine-induced hemopoietic suppression in aplastic anemia

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12 December 2020

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Impact of pre-analytical handling on bone marrow mRNA gene expression

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Prevalence and prognostic significance of allelic imbalance by single-nucleotide polymorphism analysis in low-risk myelodysplastic syndromes

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Identifiers

10.1074/JBC.M111.235028

1 reference

Europe PubMed Central

6 August 2017

1 reference

Europe PubMed Central

6 August 2017

1 reference

Europe PubMed Central

6 August 2017

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