Presenilins in memory, Alzheimer's disease, and therapy (Q35748755)

10 August 2017

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title

Presenilins in memory, Alzheimer's disease, and therapy (English)

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10 August 2017

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3

Bart De Strooper

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10 August 2017

Els Marjaux

1

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10 August 2017

Dieter Hartmann

series ordinal

2

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10 August 2017

language of work or name

English

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publication date

1 April 2004

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10 August 2017

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10 August 2017

volume

42

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10 August 2017

issue

2

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10 August 2017

page(s)

189-192

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Aph-1, Pen-2, and Nicastrin with Presenilin generate an active gamma-Secretase complex

10 August 2017

cites work

1 reference

https://api.crossref.org/works/10.1016%2FS0896-6273%2804%2900218-1

7 January 2021

Mice lacking both presenilin genes exhibit early embryonic patterning defects

1 reference

https://api.crossref.org/works/10.1016%2FS0896-6273%2804%2900218-1

7 January 2021

Deficient neurogenesis in forebrain-specific presenilin-1 knockout mice is associated with reduced clearance of hippocampal memory traces

1 reference

https://api.crossref.org/works/10.1016%2FS0896-6273%2804%2900218-1

7 January 2021

Presenilin-1 deficiency leads to loss of Cajal-Retzius neurons and cortical dysplasia similar to human type 2 lissencephaly

1 reference

https://api.crossref.org/works/10.1016%2FS0896-6273%2804%2900218-1

7 January 2021

Presenilin 2 deficiency causes a mild pulmonary phenotype and no changes in amyloid precursor protein processing but enhances the embryonic lethal phenotype of presenilin 1 deficiency

1 reference

https://api.crossref.org/works/10.1016%2FS0896-6273%2804%2900218-1

7 January 2021

Abnormal blood vessel development in mice lacking presenilin-1.

1 reference

https://api.crossref.org/works/10.1016%2FS0896-6273%2804%2900218-1

7 January 2021

Notch1 functions as a tumor suppressor in mouse skin

1 reference

https://api.crossref.org/works/10.1016%2FS0896-6273%2804%2900218-1

7 January 2021

Presenilins mutated at Asp-257 or Asp-385 restore Pen-2 expression and Nicastrin glycosylation but remain catalytically inactive in the absence of wild type Presenilin

1 reference

https://api.crossref.org/works/10.1016%2FS0896-6273%2804%2900218-1

7 January 2021

Early embryonic lethality caused by targeted disruption of the 3-hydroxy-3-methylglutaryl-CoA reductase gene

1 reference

https://api.crossref.org/works/10.1016%2FS0896-6273%2804%2900218-1

7 January 2021

Loss of presenilin function causes impairments of memory and synaptic plasticity followed by age-dependent neurodegeneration

1 reference

https://api.crossref.org/works/10.1016%2FS0896-6273%2804%2900218-1

7 January 2021

Skeletal and CNS defects in Presenilin-1-deficient mice

1 reference

https://api.crossref.org/works/10.1016%2FS0896-6273%2804%2900218-1

7 January 2021

Mesp2 initiates somite segmentation through the Notch signalling pathway.

1 reference

https://api.crossref.org/works/10.1016%2FS0896-6273%2804%2900218-1

7 January 2021

Presenilins are required for the formation of comma- and S-shaped bodies during nephrogenesis

1 reference

https://api.crossref.org/works/10.1016%2FS0896-6273%2804%2900218-1

7 January 2021

A subset of NSAIDs lower amyloidogenic Abeta42 independently of cyclooxygenase activity

1 reference

https://api.crossref.org/works/10.1016%2FS0896-6273%2804%2900218-1

7 January 2021

Presenilin 1 is required for Notch1 and DII1 expression in the paraxial mesoderm

1 reference

https://api.crossref.org/works/10.1016%2FS0896-6273%2804%2900218-1

7 January 2021

Loss of presenilin 1 is associated with enhanced beta-catenin signaling and skin tumorigenesis

1 reference

https://api.crossref.org/works/10.1016%2FS0896-6273%2804%2900218-1

7 January 2021

APP processing and synaptic plasticity in presenilin-1 conditional knockout mice

1 reference

https://api.crossref.org/works/10.1016%2FS0896-6273%2804%2900218-1

7 January 2021

10.1016/S0896-6273(04)00218-1

Identifiers

DOI

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10 August 2017

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