Inhibition of lethal inflammatory responses through the targeting of membrane-associated Toll-like receptor 4 signaling complexes with a Smad6-derived peptide. (Q35822572)

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scientific article published on 12 March 2015

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English	Inhibition of lethal inflammatory responses through the targeting of membrane-associated Toll-like receptor 4 signaling complexes with a Smad6-derived peptide.	scientific article published on 12 March 2015

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scholarly article

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2 March 2020

Inhibition of lethal inflammatory responses through the targeting of membrane-associated Toll-like receptor 4 signaling complexes with a Smad6-derived peptide (English)

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2 March 2020

toll-like receptor

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8

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2 March 2020

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Jin Seok Park

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Seok Hee Park

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2 March 2020

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Youn Sook Lee

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2 March 2020

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Su Myung Jung

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Jun Hwan Kim

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Yoe-Sik Bae

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2 March 2020

author name string

Sang-Doo Kim

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Jae Young Lee

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Kyeong Cheon Jung

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2 March 2020

Sangho Lee

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2 March 2020

Seong-Jin Kim

10

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publication date

1 May 2015

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2 March 2020

EMBO Molecular Medicine

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7

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2 March 2020

5

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2 March 2020

577-592

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2 March 2020

The roles of Pellino E3 ubiquitin ligases in immunity

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Regulation of RIP1 kinase signalling at the crossroads of inflammation and cell death

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Pellino3 targets RIP1 and regulates the pro-apoptotic effects of TNF-α

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Pellino3 ubiquitinates RIP2 and mediates Nod2-induced signaling and protective effects in colitis

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Effect of eritoran, an antagonist of MD2-TLR4, on mortality in patients with severe sepsis: the ACCESS randomized trial

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Smad6-specific recruitment of Smurf E3 ligases mediates TGF-β1-induced degradation of MyD88 in TLR4 signalling

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The ubiquitin ligase Peli1 negatively regulates T cell activation and prevents autoimmunity

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Immune unresponsiveness to secondary heterologous bacterial infection after sepsis induction is TRAIL dependent

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The pathogenesis of sepsis.

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Sepsis-induced apoptosis leads to active suppression of delayed-type hypersensitivity by CD8+ regulatory T cells through a TRAIL-dependent mechanism

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Toll-like receptor 4 modulation as a strategy to treat sepsis

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Pattern recognition receptors and inflammation

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Peli1 facilitates TRIF-dependent Toll-like receptor signaling and proinflammatory cytokine production

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Regulation of chemokine receptor by Toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis

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Pellino Proteins Contain a Cryptic FHA Domain that Mediates Interaction with Phosphorylated IRAK1

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Role of Toll-like receptors in the development of sepsis

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Role of Toll-like receptor responses for sepsis pathogenesis

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The IRAK-catalysed activation of the E3 ligase function of Pellino isoforms induces the Lys63-linked polyubiquitination of IRAK1.

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Kinase-active interleukin-1 receptor-associated kinases promote polyubiquitination and degradation of the Pellino family: direct evidence for PELLINO proteins being ubiquitin-protein isopeptide ligases

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Apoptosis and caspases regulate death and inflammation in sepsis

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Smad6 negatively regulates interleukin 1-receptor-Toll-like receptor signaling through direct interaction with the adaptor Pellino-1.

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GRKs and arrestins: regulators of migration and inflammation

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Pellino proteins are more than scaffold proteins in TLR/IL-1R signalling: a role as novel RING E3-ubiquitin-ligases

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Neutrophils and immunity: challenges and opportunities

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Cecal ligation and puncture

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12 December 2020

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inferred from PubMed ID database lookup

Expression and function of the chemokine receptors CXCR1 and CXCR2 in sepsis

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12 December 2020

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10.15252/EMMM.201404653

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Europe PubMed Central

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2 March 2020

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Europe PubMed Central

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2 March 2020

PubMed publication ID

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Europe PubMed Central

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2 March 2020

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